| Id |
Subject |
Object |
Predicate |
Lexical cue |
| T244 |
0-20 |
Sentence |
denotes |
Pulmonary conditions |
| T245 |
21-241 |
Sentence |
denotes |
It has been postulated that the neuroinvasion of the brain’s medulla oblongata region by the SARS-CoV-2 virus might impair cardiorespiratory control centers and, in that way, lead to respiratory distress and failure [4]. |
| T246 |
242-369 |
Sentence |
denotes |
The literature reports a COVID-19 patient who lost the involuntary process of breathing and suffered respiratory failure [115]. |
| T247 |
370-511 |
Sentence |
denotes |
ACE-2 receptors are expressed abundantly in the body, even in the brain, and particularly in the brain stem which controls respiration [116]. |
| T248 |
512-664 |
Sentence |
denotes |
In the event that the SARS-CoV-2 virus might enter the microcirculatory system of the brain, it may access ACE-2 receptors in the capillary endothelium. |
| T249 |
665-794 |
Sentence |
denotes |
In such a situation, the virus can cause damage to the endothelium itself, enter the brain, and cause neuronal destruction [116]. |
| T250 |
795-972 |
Sentence |
denotes |
Better understanding is urgently needed to elucidate the role of the SARS-CoV-2 virus in the brain and to risk-stratify patients who may be suffering COVID-related brain damage. |
