PMC:7417114 / 70644-71785 JSONTXT

{"target":"https://pubannotation.org/docs/sourcedb/PMC/sourceid/7417114","sourcedb":"PMC","sourceid":"7417114","source_url":"https://www.ncbi.nlm.nih.gov/pmc/7417114","text":"Among numerous other kinase inhibitors with demonstrated therapeutic potential are axitinib, which, through VEGFR-3 (vascular endothelial growth factor receptor-3) inhibition, improved lymphangiogenesis and oxygen saturation in preclinical model of aspiration pneumonia (128). Nintedanib is approved for idiopathic pulmonary fibrosis and displays anti-inflammatory activity through TNF-alpha and IL-6 inhibition (129) (130) (131). Ibrutinib exerted anti-inflammatory effects in a model of neuroinflammation-related disease (132) and mitigated acute lung inflammation in a model of pneumococcal pneumonia (133). Alvocidib also shows anti-inflammatory activity by blocking leukocyte-endothelial association by inhibiting CDK9 (134). Pazopanib exhibits antifibrotic activity through modulating inflammatory cytokines, and sunitinib inhibited bleomycin-induced pulmonary fibrosis in mice (135) (136). Sunitinib, believed to primarily work through PDGFR-mediated signaling, was also shown to suppress cytokine storm in a mouse model (137). Finally, sunitinib was shown to synergize with an anti-TNF antibody against lethal dengue infection 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