PMC:3506840 / 8875-9557 JSONTXT

{"project":"AxD_symptoms","denotations":[{"id":"T36","span":{"begin":388,"end":400},"obj":"Phenotype"},{"id":"T37","span":{"begin":659,"end":676},"obj":"Phenotype"}],"attributes":[{"id":"A36","pred":"hp_id","subj":"T36","obj":"http://purl.obolibrary.org/obo/HP_0002383"},{"id":"A37","pred":"hp_id","subj":"T37","obj":"http://purl.obolibrary.org/obo/HP_0007366"}],"text":"Our case differs, however, in that the medullary lesion was confined to a tiny portion of the dorsal region, and seems too small to be responsible for expansion of the cervicomedullary lesion, suggesting that the latter, which is reminiscent of chronic progressive neuro-Behçet’s disease, results from a different pathological process. [12]. In neuro-Behçet’s disease, subacute brainstem encephalitis, which is the most common in parenchymal involvement, shows hypertense T2 lesion with contrast enhancement. Spontaneous self-remission without therapy has been reported, although, some patients develop a slowly progressive disease with insidiously advancing brainstem atrophy [12]."}

{"project":"2_test","denotations":[{"id":"22198646-19161910-75888816","span":{"begin":337,"end":339},"obj":"19161910"},{"id":"22198646-19161910-75888817","span":{"begin":678,"end":680},"obj":"19161910"}],"text":"Our case differs, however, in that the medullary lesion was confined to a tiny portion of the dorsal region, and seems too small to be responsible for expansion of the cervicomedullary lesion, suggesting that the latter, which is reminiscent of chronic progressive neuro-Behçet’s disease, results from a different pathological process. [12]. In neuro-Behçet’s disease, subacute brainstem encephalitis, which is the most common in parenchymal involvement, shows hypertense T2 lesion with contrast enhancement. Spontaneous self-remission without therapy has been reported, although, some patients develop a slowly progressive disease with insidiously advancing brainstem atrophy [12]."}