PMC:3245220 / 1416-1933 JSONTXT

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    bionlp-st-ge-2016-uniprot

    {"project":"bionlp-st-ge-2016-uniprot","denotations":[{"id":"T3308","span":{"begin":93,"end":129},"obj":"P09603"},{"id":"T3309","span":{"begin":104,"end":129},"obj":"P04141"},{"id":"T3310","span":{"begin":131,"end":136},"obj":"P09603"},{"id":"T3311","span":{"begin":133,"end":136},"obj":"P04141"},{"id":"T3312","span":{"begin":218,"end":223},"obj":"P09603"},{"id":"T3313","span":{"begin":220,"end":223},"obj":"P04141"},{"id":"T3314","span":{"begin":474,"end":479},"obj":"P09603"},{"id":"T3315","span":{"begin":476,"end":479},"obj":"P04141"}],"namespaces":[{"prefix":"_base","uri":"http://www.uniprot.org/uniprot/"}],"text":"in blood for 24–48 hours [1]. In the absence of serum, monocytes die via apoptosis [1], [2]. Macrophage colony-stimulating factor (M-CSF) stimulates mononuclear phagocyte survival and differentiation [3]. Importantly, M-CSF-mediated cell survival and activation is associated with a variety of human diseases, including atherosclerosis, transplant vascular sclerosis and breast cancer metastasis [4], [5], [6].\nWe previously identified that NF-κB activation is important in M-CSF-induced monocyte survival [7]. In add"}

    bionlp-st-ge-2016-reference

    {"project":"bionlp-st-ge-2016-reference","denotations":[{"id":"T3526","span":{"begin":93,"end":129},"obj":"Protein"},{"id":"T3527","span":{"begin":131,"end":136},"obj":"Protein"},{"id":"T3528","span":{"begin":474,"end":479},"obj":"Protein"},{"id":"T3529","span":{"begin":218,"end":223},"obj":"Protein"}],"relations":[{"id":"R2985","pred":"equivalentTo","subj":"T3527","obj":"T3526"}],"namespaces":[{"prefix":"_base","uri":"http://bionlp.dbcls.jp/ontology/ge.owl#"}],"text":"in blood for 24–48 hours [1]. In the absence of serum, monocytes die via apoptosis [1], [2]. Macrophage colony-stimulating factor (M-CSF) stimulates mononuclear phagocyte survival and differentiation [3]. Importantly, M-CSF-mediated cell survival and activation is associated with a variety of human diseases, including atherosclerosis, transplant vascular sclerosis and breast cancer metastasis [4], [5], [6].\nWe previously identified that NF-κB activation is important in M-CSF-induced monocyte survival [7]. In add"}

    bionlp-st-ge-2016-reference-tees

    {"project":"bionlp-st-ge-2016-reference-tees","denotations":[{"id":"T4393","span":{"begin":93,"end":129},"obj":"Protein"},{"id":"T4394","span":{"begin":131,"end":136},"obj":"Protein"},{"id":"T4395","span":{"begin":218,"end":223},"obj":"Protein"},{"id":"T4396","span":{"begin":441,"end":446},"obj":"Protein"},{"id":"T4397","span":{"begin":474,"end":479},"obj":"Protein"},{"id":"T4398","span":{"begin":447,"end":457},"obj":"Positive_regulation"}],"relations":[{"id":"R3330","pred":"themeOf","subj":"T4396","obj":"T4398"}],"text":"in blood for 24–48 hours [1]. In the absence of serum, monocytes die via apoptosis [1], [2]. Macrophage colony-stimulating factor (M-CSF) stimulates mononuclear phagocyte survival and differentiation [3]. Importantly, M-CSF-mediated cell survival and activation is associated with a variety of human diseases, including atherosclerosis, transplant vascular sclerosis and breast cancer metastasis [4], [5], [6].\nWe previously identified that NF-κB activation is important in M-CSF-induced monocyte survival [7]. In add"}

    events-check-again

    {"project":"events-check-again","denotations":[{"id":"T4262","span":{"begin":93,"end":129},"obj":"Protein"},{"id":"T4263","span":{"begin":131,"end":136},"obj":"Protein"},{"id":"T4264","span":{"begin":218,"end":223},"obj":"Protein"},{"id":"T4265","span":{"begin":474,"end":479},"obj":"Protein"}],"relations":[{"id":"R3218","pred":"equivalentTo","subj":"T4263","obj":"T4262"}],"text":"in blood for 24–48 hours [1]. In the absence of serum, monocytes die via apoptosis [1], [2]. Macrophage colony-stimulating factor (M-CSF) stimulates mononuclear phagocyte survival and differentiation [3]. Importantly, M-CSF-mediated cell survival and activation is associated with a variety of human diseases, including atherosclerosis, transplant vascular sclerosis and breast cancer metastasis [4], [5], [6].\nWe previously identified that NF-κB activation is important in M-CSF-induced monocyte survival [7]. In add"}

    ICD10

    {"project":"ICD10","denotations":[{"id":"T3588","span":{"begin":320,"end":335},"obj":"http://purl.bioontology.org/ontology/ICD10/I70"}],"text":"in blood for 24–48 hours [1]. In the absence of serum, monocytes die via apoptosis [1], [2]. Macrophage colony-stimulating factor (M-CSF) stimulates mononuclear phagocyte survival and differentiation [3]. Importantly, M-CSF-mediated cell survival and activation is associated with a variety of human diseases, including atherosclerosis, transplant vascular sclerosis and breast cancer metastasis [4], [5], [6].\nWe previously identified that NF-κB activation is important in M-CSF-induced monocyte survival [7]. In add"}

    GO-CC

    {"project":"GO-CC","denotations":[{"id":"T3618","span":{"begin":233,"end":237},"obj":"http://purl.obolibrary.org/obo/GO_0005623"}],"text":"in blood for 24–48 hours [1]. In the absence of serum, monocytes die via apoptosis [1], [2]. Macrophage colony-stimulating factor (M-CSF) stimulates mononuclear phagocyte survival and differentiation [3]. Importantly, M-CSF-mediated cell survival and activation is associated with a variety of human diseases, including atherosclerosis, transplant vascular sclerosis and breast cancer metastasis [4], [5], [6].\nWe previously identified that NF-κB activation is important in M-CSF-induced monocyte survival [7]. In add"}

    GO-BP

    {"project":"GO-BP","denotations":[{"id":"T4585","span":{"begin":73,"end":82},"obj":"http://purl.obolibrary.org/obo/GO_0006915"},{"id":"T4639","span":{"begin":73,"end":82},"obj":"http://purl.obolibrary.org/obo/GO_0097194"}],"text":"in blood for 24–48 hours [1]. In the absence of serum, monocytes die via apoptosis [1], [2]. Macrophage colony-stimulating factor (M-CSF) stimulates mononuclear phagocyte survival and differentiation [3]. Importantly, M-CSF-mediated cell survival and activation is associated with a variety of human diseases, including atherosclerosis, transplant vascular sclerosis and breast cancer metastasis [4], [5], [6].\nWe previously identified that NF-κB activation is important in M-CSF-induced monocyte survival [7]. In add"}

    UBERON-AE

    {"project":"UBERON-AE","denotations":[{"id":"T961","span":{"begin":3,"end":8},"obj":"http://purl.obolibrary.org/obo/UBERON_0000178"},{"id":"T962","span":{"begin":48,"end":53},"obj":"http://purl.obolibrary.org/obo/UBERON_0001977"},{"id":"T963","span":{"begin":371,"end":377},"obj":"http://purl.obolibrary.org/obo/UBERON_0000310"}],"text":"in blood for 24–48 hours [1]. In the absence of serum, monocytes die via apoptosis [1], [2]. Macrophage colony-stimulating factor (M-CSF) stimulates mononuclear phagocyte survival and differentiation [3]. Importantly, M-CSF-mediated cell survival and activation is associated with a variety of human diseases, including atherosclerosis, transplant vascular sclerosis and breast cancer metastasis [4], [5], [6].\nWe previously identified that NF-κB activation is important in M-CSF-induced monocyte survival [7]. In add"}

    pmc-enju-pas

    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blood for 24–48 hours [1]. In the absence of serum, monocytes die via apoptosis [1], [2]. Macrophage colony-stimulating factor (M-CSF) stimulates mononuclear phagocyte survival and differentiation [3]. Importantly, M-CSF-mediated cell survival and activation is associated with a variety of human diseases, including atherosclerosis, transplant vascular sclerosis and breast cancer metastasis [4], [5], [6].\nWe previously identified that NF-κB activation is important in M-CSF-induced monocyte survival [7]. In add"}

    sentences

    {"project":"sentences","denotations":[{"id":"T969","span":{"begin":30,"end":92},"obj":"Sentence"},{"id":"T970","span":{"begin":93,"end":204},"obj":"Sentence"},{"id":"T971","span":{"begin":205,"end":410},"obj":"Sentence"},{"id":"T972","span":{"begin":411,"end":510},"obj":"Sentence"},{"id":"T12","span":{"begin":30,"end":92},"obj":"Sentence"},{"id":"T13","span":{"begin":93,"end":204},"obj":"Sentence"},{"id":"T14","span":{"begin":205,"end":410},"obj":"Sentence"},{"id":"T15","span":{"begin":411,"end":510},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"in blood for 24–48 hours [1]. In the absence of serum, monocytes die via apoptosis [1], [2]. Macrophage colony-stimulating factor (M-CSF) stimulates mononuclear phagocyte survival and differentiation [3]. Importantly, M-CSF-mediated cell survival and activation is associated with a variety of human diseases, including atherosclerosis, transplant vascular sclerosis and breast cancer metastasis [4], [5], [6].\nWe previously identified that NF-κB activation is important in M-CSF-induced monocyte survival [7]. In add"}

    2_test

    {"project":"2_test","denotations":[{"id":"22216091-355567-90609669","span":{"begin":26,"end":27},"obj":"355567"},{"id":"22216091-355567-90609670","span":{"begin":84,"end":85},"obj":"355567"},{"id":"22216091-1940344-90609671","span":{"begin":89,"end":90},"obj":"1940344"},{"id":"22216091-10473597-90609672","span":{"begin":201,"end":202},"obj":"10473597"},{"id":"22216091-15289345-90609673","span":{"begin":397,"end":398},"obj":"15289345"},{"id":"22216091-8633015-90609674","span":{"begin":402,"end":403},"obj":"8633015"},{"id":"22216091-7667279-90609675","span":{"begin":407,"end":408},"obj":"7667279"},{"id":"22216091-11804874-90609676","span":{"begin":507,"end":508},"obj":"11804874"}],"text":"in blood for 24–48 hours [1]. In the absence of serum, monocytes die via apoptosis [1], [2]. Macrophage colony-stimulating factor (M-CSF) stimulates mononuclear phagocyte survival and differentiation [3]. Importantly, M-CSF-mediated cell survival and activation is associated with a variety of human diseases, including atherosclerosis, transplant vascular sclerosis and breast cancer metastasis [4], [5], [6].\nWe previously identified that NF-κB activation is important in M-CSF-induced monocyte survival [7]. In add"}

    bionlp-st-ge-2016-spacy-parsed

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