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Modifier QTL have been mapped for numerous traits and in these studies the modified locus is typically a known gene containing a spontaneously arisen or engineered mutation with major phenotypic effects, such as hg [11,19-21]. Epistasis forms the basis of these interactions, implying that the known gene and its modifiers are members of the same biochemical or metabolic pathway. Different genetic backgrounds have been shown to modify the growth-enhancing effects of hg [15,22]. Since the primary function of Socs2 is to negatively regulate growth hormone (Gh) [23], it is likely these background effects are the result of polymorphism influencing interactions between members of the Gh signaling pathway. Thus, identification of hg modifiers has the potential to uncover novel members of metabolically important pathways or previously unknown convergences between pathways."}

{"project":"craft-ca-core-ex-dev","denotations":[{"id":"T2670","span":{"begin":0,"end":3},"obj":"SO_EXT:0000771"},{"id":"T2671","span":{"begin":84,"end":87},"obj":"SO_EXT:0000771"},{"id":"T2672","span":{"begin":103,"end":106},"obj":"SO_EXT:0000771"},{"id":"T2673","span":{"begin":169,"end":177},"obj":"SO_EXT:sequence_alteration_process"},{"id":"T2674","span":{"begin":205,"end":209},"obj":"SO_EXT:0000704"},{"id":"T2675","span":{"begin":258,"end":266},"obj":"SO_EXT:sequence_alteration_entity_or_process"},{"id":"T2676","span":{"begin":394,"end":398},"obj":"SO_EXT:0000704"},{"id":"T2677","span":{"begin":441,"end":452},"obj":"CHEBI_EXT:biochemical"},{"id":"T2678","span":{"begin":456,"end":465},"obj":"GO:0008152"},{"id":"T2679","span":{"begin":485,"end":492},"obj":"SO_EXT:0000704"},{"id":"T2680","span":{"begin":535,"end":541},"obj":"GO_EXT:biological_growth_entity_or_process"},{"id":"T2681","span":{"begin":605,"end":610},"obj":"PR_EXT:000015393"},{"id":"T2682","span":{"begin":617,"end":636},"obj":"GO_EXT:negative_regulation"},{"id":"T2683","span":{"begin":637,"end":643},"obj":"GO_EXT:biological_growth_entity_or_process"},{"id":"T2684","span":{"begin":637,"end":651},"obj":"CHEBI_GO_PR_EXT:growth_hormone"},{"id":"T2685","span":{"begin":653,"end":655},"obj":"CHEBI_GO_PR_EXT:growth_hormone"},{"id":"T2686","span":{"begin":719,"end":731},"obj":"SO_EXT:polymorphism"},{"id":"T2687","span":{"begin":780,"end":782},"obj":"CHEBI_GO_PR_EXT:growth_hormone"},{"id":"T2688","span":{"begin":780,"end":800},"obj":"GO:0060396"},{"id":"T2689","span":{"begin":885,"end":898},"obj":"GO:0008152"}],"text":"QTL which alter the phenotypic effects of another locus are referred to as modifier QTL [18]. Modifier QTL have been mapped for numerous traits and in these studies the modified locus is typically a known gene containing a spontaneously arisen or engineered mutation with major phenotypic effects, such as hg [11,19-21]. Epistasis forms the basis of these interactions, implying that the known gene and its modifiers are members of the same biochemical or metabolic pathway. Different genetic backgrounds have been shown to modify the growth-enhancing effects of hg [15,22]. Since the primary function of Socs2 is to negatively regulate growth hormone (Gh) [23], it is likely these background effects are the result of polymorphism influencing interactions between members of the Gh signaling pathway. Thus, identification of hg modifiers has the potential to uncover novel members of metabolically important pathways or previously unknown convergences between pathways."}

{"project":"craft-ca-core-dev","denotations":[{"id":"T2591","span":{"begin":0,"end":3},"obj":"SO:0000771"},{"id":"T2592","span":{"begin":84,"end":87},"obj":"SO:0000771"},{"id":"T2593","span":{"begin":103,"end":106},"obj":"SO:0000771"},{"id":"T2594","span":{"begin":205,"end":209},"obj":"SO:0000704"},{"id":"T2595","span":{"begin":394,"end":398},"obj":"SO:0000704"},{"id":"T2596","span":{"begin":456,"end":465},"obj":"GO:0008152"},{"id":"T2597","span":{"begin":485,"end":492},"obj":"SO:0000704"},{"id":"T2598","span":{"begin":605,"end":610},"obj":"PR:000015393"},{"id":"T2599","span":{"begin":780,"end":800},"obj":"GO:0060396"},{"id":"T2600","span":{"begin":885,"end":898},"obj":"GO:0008152"}],"text":"QTL which alter the phenotypic effects of another locus are referred to as modifier QTL [18]. Modifier QTL have been mapped for numerous traits and in these studies the modified locus is typically a known gene containing a spontaneously arisen or engineered mutation with major phenotypic effects, such as hg [11,19-21]. Epistasis forms the basis of these interactions, implying that the known gene and its modifiers are members of the same biochemical or metabolic pathway. Different genetic backgrounds have been shown to modify the growth-enhancing effects of hg [15,22]. Since the primary function of Socs2 is to negatively regulate growth hormone (Gh) [23], it is likely these background effects are the result of polymorphism influencing interactions between members of the Gh signaling pathway. Thus, identification of hg modifiers has the potential to uncover novel members of metabolically important pathways or previously unknown convergences between pathways."}