| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-96 |
Sentence |
denotes |
Accumulation of glycosphingolipids in human atherosclerotic plaque and unaffected aorta tissues. |
| T1 |
0-96 |
Sentence |
denotes |
Accumulation of glycosphingolipids in human atherosclerotic plaque and unaffected aorta tissues. |
| T1 |
0-96 |
Sentence |
denotes |
Accumulation of glycosphingolipids in human atherosclerotic plaque and unaffected aorta tissues. |
| TextSentencer_T2 |
97-281 |
Sentence |
denotes |
We have measured the levels of glycosphingolipids and the activity of glycosphingolipid glycosyltransferases in human aortic intima and media from patients who died of atherosclerosis. |
| T2 |
97-281 |
Sentence |
denotes |
We have measured the levels of glycosphingolipids and the activity of glycosphingolipid glycosyltransferases in human aortic intima and media from patients who died of atherosclerosis. |
| T2 |
97-281 |
Sentence |
denotes |
We have measured the levels of glycosphingolipids and the activity of glycosphingolipid glycosyltransferases in human aortic intima and media from patients who died of atherosclerosis. |
| TextSentencer_T3 |
282-422 |
Sentence |
denotes |
The effects of lactosylceramide (LacCer) and glucosylceramide (GlcCer) from plaque intima on smooth muscle cell proliferation were assessed. |
| T3 |
282-422 |
Sentence |
denotes |
The effects of lactosylceramide (LacCer) and glucosylceramide (GlcCer) from plaque intima on smooth muscle cell proliferation were assessed. |
| T3 |
282-422 |
Sentence |
denotes |
The effects of lactosylceramide (LacCer) and glucosylceramide (GlcCer) from plaque intima on smooth muscle cell proliferation were assessed. |
| TextSentencer_T4 |
423-608 |
Sentence |
denotes |
When the GlcCer data was expressed as (micrograms GlcCer/mg cholesterol and/mg total phospholipid, a 28-fold and 7-fold increase in plaque intima compared to normal intima was observed. |
| T4 |
423-608 |
Sentence |
denotes |
When the GlcCer data was expressed as (micrograms GlcCer/mg cholesterol and/mg total phospholipid, a 28-fold and 7-fold increase in plaque intima compared to normal intima was observed. |
| T4 |
423-608 |
Sentence |
denotes |
When the GlcCer data was expressed as (micrograms GlcCer/mg cholesterol and/mg total phospholipid, a 28-fold and 7-fold increase in plaque intima compared to normal intima was observed. |
| TextSentencer_T5 |
609-716 |
Sentence |
denotes |
Similarly, the level of LacCer was elevated 5-fold and 4-fold, respectively, compared to unaffected intima. |
| T5 |
609-716 |
Sentence |
denotes |
Similarly, the level of LacCer was elevated 5-fold and 4-fold, respectively, compared to unaffected intima. |
| T5 |
609-716 |
Sentence |
denotes |
Similarly, the level of LacCer was elevated 5-fold and 4-fold, respectively, compared to unaffected intima. |
| TextSentencer_T6 |
717-855 |
Sentence |
denotes |
The activity of UDP-GlcCer: ceramide beta 1-->4 glucosyltransferase (GlcT-1) was similar in unaffected tissue, fatty streaks, and plaques. |
| T6 |
717-855 |
Sentence |
denotes |
The activity of UDP-GlcCer: ceramide beta 1-->4 glucosyltransferase (GlcT-1) was similar in unaffected tissue, fatty streaks, and plaques. |
| T6 |
717-855 |
Sentence |
denotes |
The activity of UDP-GlcCer: ceramide beta 1-->4 glucosyltransferase (GlcT-1) was similar in unaffected tissue, fatty streaks, and plaques. |
| TextSentencer_T7 |
856-895 |
Sentence |
denotes |
However, the activity of UDP-galactose: |
| T7 |
856-895 |
Sentence |
denotes |
However, the activity of UDP-galactose: |
| T7 |
856-895 |
Sentence |
denotes |
However, the activity of UDP-galactose: |
| TextSentencer_T8 |
896-1013 |
Sentence |
denotes |
GlcCer, beta 1-->4 galactosyltransferase (GalT-2) activity was moderately higher in plaque than in unaffected tissue. |
| T8 |
896-1013 |
Sentence |
denotes |
GlcCer, beta 1-->4 galactosyltransferase (GalT-2) activity was moderately higher in plaque than in unaffected tissue. |
| T8 |
896-1013 |
Sentence |
denotes |
GlcCer, beta 1-->4 galactosyltransferase (GalT-2) activity was moderately higher in plaque than in unaffected tissue. |
| TextSentencer_T9 |
1014-1355 |
Sentence |
denotes |
LacCer, but not GlcCer derived from plaque intima exerted a approximately 2.8-fold increase in the proliferation of human aortic smooth muscle cells grown in tissue culture compared to control presumably due to a marked increase in LacCer molecular species containing C16:0, C22:1, and C24:0 fatty acids in plaque intima compared to control. |
| T9 |
1014-1355 |
Sentence |
denotes |
LacCer, but not GlcCer derived from plaque intima exerted a approximately 2.8-fold increase in the proliferation of human aortic smooth muscle cells grown in tissue culture compared to control presumably due to a marked increase in LacCer molecular species containing C16:0, C22:1, and C24:0 fatty acids in plaque intima compared to control. |
| T9 |
1014-1355 |
Sentence |
denotes |
LacCer, but not GlcCer derived from plaque intima exerted a approximately 2.8-fold increase in the proliferation of human aortic smooth muscle cells grown in tissue culture compared to control presumably due to a marked increase in LacCer molecular species containing C16:0, C22:1, and C24:0 fatty acids in plaque intima compared to control. |
| TextSentencer_T10 |
1356-1536 |
Sentence |
denotes |
In sum, our findings provide an interesting and novel pathogenic mechanism of lactosylceramide mediated plaque formation via stimulation of aortic smooth muscle cell proliferation. |
| T10 |
1356-1536 |
Sentence |
denotes |
In sum, our findings provide an interesting and novel pathogenic mechanism of lactosylceramide mediated plaque formation via stimulation of aortic smooth muscle cell proliferation. |
| T10 |
1356-1536 |
Sentence |
denotes |
In sum, our findings provide an interesting and novel pathogenic mechanism of lactosylceramide mediated plaque formation via stimulation of aortic smooth muscle cell proliferation. |