| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-78 |
Sentence |
denotes |
Prevalence of apolipoprotein E phenotypes in ischemic cerebrovascular disease. |
| T1 |
0-78 |
Sentence |
denotes |
Prevalence of apolipoprotein E phenotypes in ischemic cerebrovascular disease. |
| TextSentencer_T2 |
79-100 |
Sentence |
denotes |
A case-control study. |
| T2 |
79-100 |
Sentence |
denotes |
A case-control study. |
| TextSentencer_T3 |
101-124 |
Sentence |
denotes |
BACKGROUND AND PURPOSE: |
| T3 |
101-124 |
Sentence |
denotes |
BACKGROUND AND PURPOSE: |
| TextSentencer_T4 |
125-218 |
Sentence |
denotes |
Apolipoprotein E polymorphism may influence the early development of coronary artery disease. |
| T4 |
125-218 |
Sentence |
denotes |
Apolipoprotein E polymorphism may influence the early development of coronary artery disease. |
| TextSentencer_T5 |
219-307 |
Sentence |
denotes |
We investigated the putative role of apolipoprotein E phenotypes in cerebral infarction. |
| T5 |
219-307 |
Sentence |
denotes |
We investigated the putative role of apolipoprotein E phenotypes in cerebral infarction. |
| TextSentencer_T6 |
308-316 |
Sentence |
denotes |
METHODS: |
| T6 |
308-316 |
Sentence |
denotes |
METHODS: |
| TextSentencer_T7 |
317-581 |
Sentence |
denotes |
The apolipoprotein E phenotypes of 69 patients (mean +/- SD age, 72 +/- 11 years) who had suffered completed stroke or a transient ischemic attack and 68 sex- and age-matched control subjects free of cerebrovascular disease were determined by isoelectric focusing. |
| T7 |
317-581 |
Sentence |
denotes |
The apolipoprotein E phenotypes of 69 patients (mean +/- SD age, 72 +/- 11 years) who had suffered completed stroke or a transient ischemic attack and 68 sex- and age-matched control subjects free of cerebrovascular disease were determined by isoelectric focusing. |
| TextSentencer_T8 |
582-729 |
Sentence |
denotes |
The relative frequency of the apolipoprotein E phenotypes in the general population was estimated in 498 healthy blood donors (mean age, 37 years). |
| T8 |
582-729 |
Sentence |
denotes |
The relative frequency of the apolipoprotein E phenotypes in the general population was estimated in 498 healthy blood donors (mean age, 37 years). |
| TextSentencer_T9 |
730-738 |
Sentence |
denotes |
RESULTS: |
| T9 |
730-738 |
Sentence |
denotes |
RESULTS: |
| TextSentencer_T10 |
739-893 |
Sentence |
denotes |
The prevalences of hypertension, diabetes mellitus, obesity, and intermittent claudication were significantly higher in patients than in control subjects. |
| T10 |
739-893 |
Sentence |
denotes |
The prevalences of hypertension, diabetes mellitus, obesity, and intermittent claudication were significantly higher in patients than in control subjects. |
| TextSentencer_T11 |
894-1066 |
Sentence |
denotes |
Serum lipid and apolipoprotein B concentrations and the composition of very low density lipoproteins were not significantly different between patients and control subjects. |
| T11 |
894-1066 |
Sentence |
denotes |
Serum lipid and apolipoprotein B concentrations and the composition of very low density lipoproteins were not significantly different between patients and control subjects. |
| TextSentencer_T12 |
1067-1136 |
Sentence |
denotes |
Apolipoprotein A-I and E levels were significantly lower in patients. |
| T12 |
1067-1136 |
Sentence |
denotes |
Apolipoprotein A-I and E levels were significantly lower in patients. |
| TextSentencer_T13 |
1137-1370 |
Sentence |
denotes |
Cholesterol levels were higher in male patients than in male control subjects (5.10 +/- 1.46 versus 4.41 +/- 0.80 mmol/L; p = 0.036), and the ratio of apolipoprotein A-I to B was lower (0.77 +/- 0.29 versus 1.03 +/- 0.37; p < 0.001). |
| T13 |
1137-1370 |
Sentence |
denotes |
Cholesterol levels were higher in male patients than in male control subjects (5.10 +/- 1.46 versus 4.41 +/- 0.80 mmol/L; p = 0.036), and the ratio of apolipoprotein A-I to B was lower (0.77 +/- 0.29 versus 1.03 +/- 0.37; p < 0.001). |
| TextSentencer_T14 |
1371-1511 |
Sentence |
denotes |
The E3/E3 phenotype was more frequent in control subjects (85%) than in patients (72.5%; p < 0.05) and healthy blood donors (64%; p < 0.02). |
| T14 |
1371-1511 |
Sentence |
denotes |
The E3/E3 phenotype was more frequent in control subjects (85%) than in patients (72.5%; p < 0.05) and healthy blood donors (64%; p < 0.02). |
| TextSentencer_T15 |
1512-1612 |
Sentence |
denotes |
The E3/E2 phenotype was more frequent in patients (10.1%) than in control subjects (1.4%; p < 0.05). |
| T15 |
1512-1612 |
Sentence |
denotes |
The E3/E2 phenotype was more frequent in patients (10.1%) than in control subjects (1.4%; p < 0.05). |
| TextSentencer_T16 |
1613-1858 |
Sentence |
denotes |
A stepwise logistic regression showed that the presence of stroke was significantly related to high blood pressure (p < 0.0001), low apo E levels (p < 0.008), obesity (p < 0.041), the apo E phenotype (p < 0.05), and diabetes mellitus (p < 0.05). |
| T16 |
1613-1858 |
Sentence |
denotes |
A stepwise logistic regression showed that the presence of stroke was significantly related to high blood pressure (p < 0.0001), low apo E levels (p < 0.008), obesity (p < 0.041), the apo E phenotype (p < 0.05), and diabetes mellitus (p < 0.05). |
| TextSentencer_T17 |
1859-1871 |
Sentence |
denotes |
CONCLUSIONS: |
| T17 |
1859-1871 |
Sentence |
denotes |
CONCLUSIONS: |
| TextSentencer_T18 |
1872-2072 |
Sentence |
denotes |
The E3/E3 phenotype may protect against early vascular morbidity, and the epsilon 2 gene may be a risk factor for cerebrovascular morbidity, possibly related to diabetes, hypertension, and/or obesity. |
| T18 |
1872-2072 |
Sentence |
denotes |
The E3/E3 phenotype may protect against early vascular morbidity, and the epsilon 2 gene may be a risk factor for cerebrovascular morbidity, possibly related to diabetes, hypertension, and/or obesity. |