| Id |
Subject |
Object |
Predicate |
Lexical cue |
| T1 |
0-177 |
Sentence |
denotes |
Formation of 6-oxoprostaglandin F1 alpha, 6,15-dioxoprostaglandin F1 alpha, and monohydroxyicosatetraenoic acids from arachidonic acid by fetal calf aorta and ductus arteriosus. |
| T1 |
0-177 |
Sentence |
denotes |
Formation of 6-oxoprostaglandin F1 alpha, 6,15-dioxoprostaglandin F1 alpha, and monohydroxyicosatetraenoic acids from arachidonic acid by fetal calf aorta and ductus arteriosus. |
| T2 |
178-483 |
Sentence |
denotes |
Particulate fractions and slices from fetal calf aorta convert arachidonic acid to 6-oxoprostaglandin F1 alpha (6-oxoPGF1 alpha), 6,15-dioxoPGF1 alpha, 12-hydroxy-5,8,10-heptadecatrienoic acid, 11-hydroxy-5,8,12,14-icosatetraenoic acid (11h-20:4), and 15-hydroxy-5,8,11,13-icosatetraenoic acid (15h-20:4). |
| T2 |
178-483 |
Sentence |
denotes |
Particulate fractions and slices from fetal calf aorta convert arachidonic acid to 6-oxoprostaglandin F1 alpha (6-oxoPGF1 alpha), 6,15-dioxoPGF1 alpha, 12-hydroxy-5,8,10-heptadecatrienoic acid, 11-hydroxy-5,8,12,14-icosatetraenoic acid (11h-20:4), and 15-hydroxy-5,8,11,13-icosatetraenoic acid (15h-20:4). |
| T3 |
484-588 |
Sentence |
denotes |
In some cases, small amounts of 12-hydroxy-5,8,10,14-icosatetraenoic acid (12h-20:4) were also detected. |
| T3 |
484-588 |
Sentence |
denotes |
In some cases, small amounts of 12-hydroxy-5,8,10,14-icosatetraenoic acid (12h-20:4) were also detected. |
| T4 |
589-749 |
Sentence |
denotes |
The products were all identified by gas chromatography-mass spectrometry after purification by normal phase and argentation high pressure liquid chromatography. |
| T4 |
589-749 |
Sentence |
denotes |
The products were all identified by gas chromatography-mass spectrometry after purification by normal phase and argentation high pressure liquid chromatography. |
| T5 |
750-959 |
Sentence |
denotes |
Both 11h-20:4 and 15h-20:4 appeared to be formed by prostaglandin endoperoxide synthetase rather than by lipoxygenases, since their formation was inhibited by indomethacin but not by nordihydroguaiaretic acid. |
| T5 |
750-959 |
Sentence |
denotes |
Both 11h-20:4 and 15h-20:4 appeared to be formed by prostaglandin endoperoxide synthetase rather than by lipoxygenases, since their formation was inhibited by indomethacin but not by nordihydroguaiaretic acid. |
| T6 |
960-1087 |
Sentence |
denotes |
The formation of 12h-20:4, on the other hand, was stimulated by indomethacin, probably due to increased substrate availability. |
| T6 |
960-1087 |
Sentence |
denotes |
The formation of 12h-20:4, on the other hand, was stimulated by indomethacin, probably due to increased substrate availability. |
| T7 |
1088-1172 |
Sentence |
denotes |
The formation of hydroxyicosatetraenoic acids was markedly stimulated by adrenaline. |
| T7 |
1088-1172 |
Sentence |
denotes |
The formation of hydroxyicosatetraenoic acids was markedly stimulated by adrenaline. |
| T8 |
1173-1378 |
Sentence |
denotes |
Substantial amounts of 6,15-dioxoPGF1 alpha were formed from arachidonic acid by particulate fractions from fetal calf blood vessels, especially in the presence of relatively high substrate concentrations. |
| T8 |
1173-1378 |
Sentence |
denotes |
Substantial amounts of 6,15-dioxoPGF1 alpha were formed from arachidonic acid by particulate fractions from fetal calf blood vessels, especially in the presence of relatively high substrate concentrations. |
| T9 |
1379-1498 |
Sentence |
denotes |
The formation of this product was stimulated by methemoglobin and inhibited by adrenaline, glutathione, and tryptophan. |
| T9 |
1379-1498 |
Sentence |
denotes |
The formation of this product was stimulated by methemoglobin and inhibited by adrenaline, glutathione, and tryptophan. |
| T10 |
1499-1723 |
Sentence |
denotes |
It would appear that particulate fractions from fetal calf aorta convert arachidonic acid to 15-hydroperoxyPGI2, which can either be reduced in the presence of various cofactors to form PGI2 or dehydrated to give 15-oxoPGI2. |
| T10 |
1499-1723 |
Sentence |
denotes |
It would appear that particulate fractions from fetal calf aorta convert arachidonic acid to 15-hydroperoxyPGI2, which can either be reduced in the presence of various cofactors to form PGI2 or dehydrated to give 15-oxoPGI2. |
| T11 |
1724-1912 |
Sentence |
denotes |
The formation of hydroperoxides from arachidonic acid could be an important factor in regulating PGI2 synthesis in aorta, since PGI2 synthetase is strongly inhibited by such intermediates. |
| T11 |
1724-1912 |
Sentence |
denotes |
The formation of hydroperoxides from arachidonic acid could be an important factor in regulating PGI2 synthesis in aorta, since PGI2 synthetase is strongly inhibited by such intermediates. |