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PubMed:6799518 JSONTXT 41 Projects

Annnotations TAB TSV DIC JSON TextAE Lectin_function IAV-Glycan

Id Subject Object Predicate Lexical cue
T1 0-50 Sentence denotes Arachidonic acid metabolism in isolated rat aorta.
T1 0-50 Sentence denotes Arachidonic acid metabolism in isolated rat aorta.
T2 51-132 Sentence denotes Dependence of prostacyclin biosynthesis on extracellular potassium concentration.
T2 51-132 Sentence denotes Dependence of prostacyclin biosynthesis on extracellular potassium concentration.
T3 133-410 Sentence denotes Slices of rat aorta were incubated in Krebs-Ringer bicarbonate buffer for measurements of immunoreactive 6-ketoprostaglandin F1 alpha, thromboxane (TX) B2, prostaglandin (PG)E2, and PGF2 alpha, and in Tris buffer (pH 9.3) for determination of prostacyclin (PGI2)-like activity.
T3 133-410 Sentence denotes Slices of rat aorta were incubated in Krebs-Ringer bicarbonate buffer for measurements of immunoreactive 6-ketoprostaglandin F1 alpha, thromboxane (TX) B2, prostaglandin (PG)E2, and PGF2 alpha, and in Tris buffer (pH 9.3) for determination of prostacyclin (PGI2)-like activity.
T4 411-505 Sentence denotes No significant generation of TXB2, PGE2, or PGF2 alpha by rat aortic tissue could be detected.
T4 411-505 Sentence denotes No significant generation of TXB2, PGE2, or PGF2 alpha by rat aortic tissue could be detected.
T5 506-650 Sentence denotes The time-dependent release of 6-keto-PGF1 alpha Krebs-Ringer bicarbonate buffer closely correlated with PGI2 generation in alkaline Tris buffer.
T5 506-650 Sentence denotes The time-dependent release of 6-keto-PGF1 alpha Krebs-Ringer bicarbonate buffer closely correlated with PGI2 generation in alkaline Tris buffer.
T6 651-924 Sentence denotes During a 30-min incubation period, 6-keto-PGF1 alpha, release was 79.8 +/- 3.3 pmol/mg at a buffer potassium concentration of 3.9 mmol/liter and significantly increased by 23% to 98.3 +/- 8.5 pmol/mg (P less than 0.025) in the absence of potassium in the incubation medium.
T6 651-924 Sentence denotes During a 30-min incubation period, 6-keto-PGF1 alpha, release was 79.8 +/- 3.3 pmol/mg at a buffer potassium concentration of 3.9 mmol/liter and significantly increased by 23% to 98.3 +/- 8.5 pmol/mg (P less than 0.025) in the absence of potassium in the incubation medium.
T7 925-1091 Sentence denotes A smaller decrease in buffer potassium concentration to 2.1 mmol/liter and an increase to 8.8 mmol/liter did not significantly alter aortic 6-keto-PGF1 alpha release.
T7 925-1091 Sentence denotes A smaller decrease in buffer potassium concentration to 2.1 mmol/liter and an increase to 8.8 mmol/liter did not significantly alter aortic 6-keto-PGF1 alpha release.
T8 1092-1302 Sentence denotes Changes in the incubation buffer sodium concentration from 144 mmol/liter to either 138 or 150 mmol/liter at a constant potassium concentration of 3.9 mmol/liter did not alter the recovery of 6-keto-PGF1 alpha.
T8 1092-1302 Sentence denotes Changes in the incubation buffer sodium concentration from 144 mmol/liter to either 138 or 150 mmol/liter at a constant potassium concentration of 3.9 mmol/liter did not alter the recovery of 6-keto-PGF1 alpha.
T9 1303-1416 Sentence denotes Our results support the concept that PGI2 is the predominant product of arachidonic acid metabolism in rat aorta.
T9 1303-1416 Sentence denotes Our results support the concept that PGI2 is the predominant product of arachidonic acid metabolism in rat aorta.
T10 1417-1536 Sentence denotes They further show that PGI2 can be recovered quantitatively as 6-keto-PGF1 alpha under the present in vitro conditions.
T10 1417-1536 Sentence denotes They further show that PGI2 can be recovered quantitatively as 6-keto-PGF1 alpha under the present in vitro conditions.
T11 1537-1691 Sentence denotes In addition, this in vitro study points to the potassium ion as a modulator of vascular PGI2 synthesis with a stimulation at low potassium concentrations.
T11 1537-1691 Sentence denotes In addition, this in vitro study points to the potassium ion as a modulator of vascular PGI2 synthesis with a stimulation at low potassium concentrations.