| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-77 |
Sentence |
denotes |
Identifying immune mechanisms mediating the hypertension during preeclampsia. |
| T1 |
0-77 |
Sentence |
denotes |
Identifying immune mechanisms mediating the hypertension during preeclampsia. |
| TextSentencer_T2 |
78-242 |
Sentence |
denotes |
Preeclampsia (PE) is a pregnancy-associated disorder that affects 5-8% of pregnancies and is a major cause of maternal, fetal, and neonatal morbidity and mortality. |
| T2 |
78-242 |
Sentence |
denotes |
Preeclampsia (PE) is a pregnancy-associated disorder that affects 5-8% of pregnancies and is a major cause of maternal, fetal, and neonatal morbidity and mortality. |
| TextSentencer_T3 |
243-442 |
Sentence |
denotes |
Hallmark characteristics of PE are new onset hypertension after 20 wk gestation with or without proteinuria, chronic immune activation, fetal growth restriction, and maternal endothelial dysfunction. |
| T3 |
243-442 |
Sentence |
denotes |
Hallmark characteristics of PE are new onset hypertension after 20 wk gestation with or without proteinuria, chronic immune activation, fetal growth restriction, and maternal endothelial dysfunction. |
| TextSentencer_T4 |
443-543 |
Sentence |
denotes |
However, the pathophysiological mechanisms that lead to the development of PE are poorly understood. |
| T4 |
443-543 |
Sentence |
denotes |
However, the pathophysiological mechanisms that lead to the development of PE are poorly understood. |
| TextSentencer_T5 |
544-757 |
Sentence |
denotes |
Recent data from studies of both clinical and animal models demonstrate an imbalance in the subpopulations of CD4+ T cells and a role for these cells as mediators of inflammation and hypertension during pregnancy. |
| T5 |
544-757 |
Sentence |
denotes |
Recent data from studies of both clinical and animal models demonstrate an imbalance in the subpopulations of CD4+ T cells and a role for these cells as mediators of inflammation and hypertension during pregnancy. |
| TextSentencer_T6 |
758-945 |
Sentence |
denotes |
Specifically, it has been proposed that the imbalance between two CD4+ T cell subtypes, regulatory T cells (Tregs) and T-helper 17 cells (Th17s), is involved in the pathophysiology of PE. |
| T6 |
758-945 |
Sentence |
denotes |
Specifically, it has been proposed that the imbalance between two CD4+ T cell subtypes, regulatory T cells (Tregs) and T-helper 17 cells (Th17s), is involved in the pathophysiology of PE. |
| TextSentencer_T7 |
946-1133 |
Sentence |
denotes |
Studies from our laboratory highlighting how this imbalance contributes to vasoactive factors, endothelial dysfunction, and hypertension during pregnancy will be discussed in this review. |
| T7 |
946-1133 |
Sentence |
denotes |
Studies from our laboratory highlighting how this imbalance contributes to vasoactive factors, endothelial dysfunction, and hypertension during pregnancy will be discussed in this review. |
| TextSentencer_T8 |
1134-1309 |
Sentence |
denotes |
Therefore, the purpose of this review is to highlight hypertensive mechanisms stimulated by inflammatory factors in response to placental ischemia, thereby elucidating a role. |
| T8 |
1134-1309 |
Sentence |
denotes |
Therefore, the purpose of this review is to highlight hypertensive mechanisms stimulated by inflammatory factors in response to placental ischemia, thereby elucidating a role. |