| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-89 |
Sentence |
denotes |
Bacterial capsular polysaccharide prevents the onset of asthma through T-cell activation. |
| T1 |
0-89 |
Sentence |
denotes |
Bacterial capsular polysaccharide prevents the onset of asthma through T-cell activation. |
| T1 |
0-89 |
Sentence |
denotes |
Bacterial capsular polysaccharide prevents the onset of asthma through T-cell activation. |
| TextSentencer_T2 |
90-244 |
Sentence |
denotes |
Over the last four decades, increases in the incidence of immune-mediated diseases in the Western world have been linked to changes in microbial exposure. |
| T2 |
90-244 |
Sentence |
denotes |
Over the last four decades, increases in the incidence of immune-mediated diseases in the Western world have been linked to changes in microbial exposure. |
| T2 |
90-244 |
Sentence |
denotes |
Over the last four decades, increases in the incidence of immune-mediated diseases in the Western world have been linked to changes in microbial exposure. |
| TextSentencer_T3 |
245-517 |
Sentence |
denotes |
It is becoming increasingly clear that the normal microbiota in the gut can profoundly alter susceptibility to a wide range of diseases, such as asthma, in which immune homeostasis is disrupted, yet the mechanisms governing this microbial influence remains poorly defined. |
| T3 |
245-517 |
Sentence |
denotes |
It is becoming increasingly clear that the normal microbiota in the gut can profoundly alter susceptibility to a wide range of diseases, such as asthma, in which immune homeostasis is disrupted, yet the mechanisms governing this microbial influence remains poorly defined. |
| T3 |
245-517 |
Sentence |
denotes |
It is becoming increasingly clear that the normal microbiota in the gut can profoundly alter susceptibility to a wide range of diseases, such as asthma, in which immune homeostasis is disrupted, yet the mechanisms governing this microbial influence remains poorly defined. |
| TextSentencer_T4 |
518-741 |
Sentence |
denotes |
In this study, we show that gastrointestinal exposure to PSA, a capsular polysaccharide derived from the commensal bacterium Bacteroides fragilis, significantly limits susceptibility to the induction of experimental asthma. |
| T4 |
518-741 |
Sentence |
denotes |
In this study, we show that gastrointestinal exposure to PSA, a capsular polysaccharide derived from the commensal bacterium Bacteroides fragilis, significantly limits susceptibility to the induction of experimental asthma. |
| T4 |
518-741 |
Sentence |
denotes |
In this study, we show that gastrointestinal exposure to PSA, a capsular polysaccharide derived from the commensal bacterium Bacteroides fragilis, significantly limits susceptibility to the induction of experimental asthma. |
| TextSentencer_T5 |
742-936 |
Sentence |
denotes |
We report that direct treatment of mice with PSA generates protection from asthma, and this effect can be given to a naïve recipient by adoptive transfer of CD4(+) T cells from PSA-exposed mice. |
| T5 |
742-936 |
Sentence |
denotes |
We report that direct treatment of mice with PSA generates protection from asthma, and this effect can be given to a naïve recipient by adoptive transfer of CD4(+) T cells from PSA-exposed mice. |
| T5 |
742-936 |
Sentence |
denotes |
We report that direct treatment of mice with PSA generates protection from asthma, and this effect can be given to a naïve recipient by adoptive transfer of CD4(+) T cells from PSA-exposed mice. |
| TextSentencer_T6 |
937-1131 |
Sentence |
denotes |
Remarkably, we found that these PSA-induced T cells are not canonical FoxP3(+) regulatory T cells, but that they potently inhibit both Th1 and Th2 models of asthma in an IL-10-dependent fashion. |
| T6 |
937-1131 |
Sentence |
denotes |
Remarkably, we found that these PSA-induced T cells are not canonical FoxP3(+) regulatory T cells, but that they potently inhibit both Th1 and Th2 models of asthma in an IL-10-dependent fashion. |
| T6 |
937-1131 |
Sentence |
denotes |
Remarkably, we found that these PSA-induced T cells are not canonical FoxP3(+) regulatory T cells, but that they potently inhibit both Th1 and Th2 models of asthma in an IL-10-dependent fashion. |
| TextSentencer_T7 |
1132-1399 |
Sentence |
denotes |
These findings reveal that bacterial polysaccharides link the microbiota with the peripheral immune system by activating CD4(+)Foxp3(-) T cells upon exposure in the gut, and they facilitate resistance to unnecessary inflammatory responses via the production of IL-10. |
| T7 |
1132-1399 |
Sentence |
denotes |
These findings reveal that bacterial polysaccharides link the microbiota with the peripheral immune system by activating CD4(+)Foxp3(-) T cells upon exposure in the gut, and they facilitate resistance to unnecessary inflammatory responses via the production of IL-10. |
| T7 |
1132-1399 |
Sentence |
denotes |
These findings reveal that bacterial polysaccharides link the microbiota with the peripheral immune system by activating CD4(+)Foxp3(-) T cells upon exposure in the gut, and they facilitate resistance to unnecessary inflammatory responses via the production of IL-10. |