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Id Subject Object Predicate Lexical cue
TextSentencer_T1 0-59 Sentence denotes Biosynthesis of the major brain gangliosides GD1a and GT1b.
T1 0-59 Sentence denotes Biosynthesis of the major brain gangliosides GD1a and GT1b.
T1 0-59 Sentence denotes Biosynthesis of the major brain gangliosides GD1a and GT1b.
TextSentencer_T2 60-148 Sentence denotes Gangliosides-sialylated glycosphingolipids-are the major glycoconjugates of nerve cells.
T2 60-148 Sentence denotes Gangliosides-sialylated glycosphingolipids-are the major glycoconjugates of nerve cells.
T2 60-148 Sentence denotes Gangliosides-sialylated glycosphingolipids-are the major glycoconjugates of nerve cells.
TextSentencer_T3 149-263 Sentence denotes The same four structures-GM1, GD1a, GD1b and GT1b-comprise the great majority of gangliosides in mammalian brains.
T3 149-263 Sentence denotes The same four structures-GM1, GD1a, GD1b and GT1b-comprise the great majority of gangliosides in mammalian brains.
T3 149-263 Sentence denotes The same four structures-GM1, GD1a, GD1b and GT1b-comprise the great majority of gangliosides in mammalian brains.
TextSentencer_T4 264-495 Sentence denotes They share a common tetrasaccharide core (Galβ1-3GalNAcβ1-4Galβ1-4Glcβ1-1'Cer) with one or two sialic acids on the internal galactose and zero (GM1 and GD1b) or one (GD1a and GT1b) α2-3-linked sialic acid on the terminal galactose.
T4 264-495 Sentence denotes They share a common tetrasaccharide core (Galβ1-3GalNAcβ1-4Galβ1-4Glcβ1-1'Cer) with one or two sialic acids on the internal galactose and zero (GM1 and GD1b) or one (GD1a and GT1b) α2-3-linked sialic acid on the terminal galactose.
T4 264-495 Sentence denotes They share a common tetrasaccharide core (Galβ1-3GalNAcβ1-4Galβ1-4Glcβ1-1'Cer) with one or two sialic acids on the internal galactose and zero (GM1 and GD1b) or one (GD1a and GT1b) α2-3-linked sialic acid on the terminal galactose.
TextSentencer_T5 496-660 Sentence denotes Whereas the genes responsible for the sialylation of the internal galactose are known, those responsible for terminal sialylation have not been established in vivo.
T5 496-660 Sentence denotes Whereas the genes responsible for the sialylation of the internal galactose are known, those responsible for terminal sialylation have not been established in vivo.
T5 496-660 Sentence denotes Whereas the genes responsible for the sialylation of the internal galactose are known, those responsible for terminal sialylation have not been established in vivo.
TextSentencer_T6 661-787 Sentence denotes We report that St3gal2 and St3gal3 are responsible for nearly all the terminal sialylation of brain gangliosides in the mouse.
T6 661-787 Sentence denotes We report that St3gal2 and St3gal3 are responsible for nearly all the terminal sialylation of brain gangliosides in the mouse.
T6 661-787 Sentence denotes We report that St3gal2 and St3gal3 are responsible for nearly all the terminal sialylation of brain gangliosides in the mouse.
TextSentencer_T7 788-1009 Sentence denotes When brain ganglioside expression was analyzed in adult St3gal1-, St3gal2-, St3gal3- and St3gal4-null mice, only St3gal2-null mice differed significantly from wild type, expressing half the normal amount of GD1a and GT1b.
T7 788-1009 Sentence denotes When brain ganglioside expression was analyzed in adult St3gal1-, St3gal2-, St3gal3- and St3gal4-null mice, only St3gal2-null mice differed significantly from wild type, expressing half the normal amount of GD1a and GT1b.
T7 788-1009 Sentence denotes When brain ganglioside expression was analyzed in adult St3gal1-, St3gal2-, St3gal3- and St3gal4-null mice, only St3gal2-null mice differed significantly from wild type, expressing half the normal amount of GD1a and GT1b.
TextSentencer_T8 1010-1171 Sentence denotes St3gal1/2-double-null mice were no different than St3gal2-single-null mice; however, St3gal2/3-double-null mice were >95% depleted in gangliosides GD1a and GT1b.
T8 1010-1171 Sentence denotes St3gal1/2-double-null mice were no different than St3gal2-single-null mice; however, St3gal2/3-double-null mice were >95% depleted in gangliosides GD1a and GT1b.
T8 1010-1171 Sentence denotes St3gal1/2-double-null mice were no different than St3gal2-single-null mice; however, St3gal2/3-double-null mice were >95% depleted in gangliosides GD1a and GT1b.
TextSentencer_T9 1172-1367 Sentence denotes Total ganglioside expression (lipid-bound sialic acid) in the brains of St3gal2/3-double-null mice was equivalent to that in wild-type mice, whereas total protein sialylation was reduced by half.
T9 1172-1367 Sentence denotes Total ganglioside expression (lipid-bound sialic acid) in the brains of St3gal2/3-double-null mice was equivalent to that in wild-type mice, whereas total protein sialylation was reduced by half.
T9 1172-1367 Sentence denotes Total ganglioside expression (lipid-bound sialic acid) in the brains of St3gal2/3-double-null mice was equivalent to that in wild-type mice, whereas total protein sialylation was reduced by half.
TextSentencer_T10 1368-1428 Sentence denotes St3gal2/3-double-null mice were small, weak and short lived.
T10 1368-1428 Sentence denotes St3gal2/3-double-null mice were small, weak and short lived.
T10 1368-1428 Sentence denotes St3gal2/3-double-null mice were small, weak and short lived.
TextSentencer_T11 1429-1525 Sentence denotes They were half the weight of wild-type mice at weaning and displayed early hindlimb dysreflexia.
T11 1429-1525 Sentence denotes They were half the weight of wild-type mice at weaning and displayed early hindlimb dysreflexia.
T11 1429-1525 Sentence denotes They were half the weight of wild-type mice at weaning and displayed early hindlimb dysreflexia.
TextSentencer_T12 1526-1763 Sentence denotes We conclude that the St3gal2 and St3gal3 gene products (ST3Gal-II and ST3Gal-III sialyltransferases) are largely responsible for ganglioside terminal α2-3 sialylation in the brain, synthesizing the major brain gangliosides GD1a and GT1b.
T12 1526-1763 Sentence denotes We conclude that the St3gal2 and St3gal3 gene products (ST3Gal-II and ST3Gal-III sialyltransferases) are largely responsible for ganglioside terminal α2-3 sialylation in the brain, synthesizing the major brain gangliosides GD1a and GT1b.
T12 1526-1763 Sentence denotes We conclude that the St3gal2 and St3gal3 gene products (ST3Gal-II and ST3Gal-III sialyltransferases) are largely responsible for ganglioside terminal α2-3 sialylation in the brain, synthesizing the major brain gangliosides GD1a and GT1b.