| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-84 |
Sentence |
denotes |
Glucose/galactose malabsorption caused by a defect in the Na+/glucose cotransporter. |
| T1 |
0-84 |
Sentence |
denotes |
Glucose/galactose malabsorption caused by a defect in the Na+/glucose cotransporter. |
| TextSentencer_T2 |
85-300 |
Sentence |
denotes |
Glucose/galactose malabsorption (GGM) is an autosomal recessive disease manifesting within the first weeks of life and characterized by a selective failure to absorb dietary glucose and galactose from the intestine. |
| T2 |
85-300 |
Sentence |
denotes |
Glucose/galactose malabsorption (GGM) is an autosomal recessive disease manifesting within the first weeks of life and characterized by a selective failure to absorb dietary glucose and galactose from the intestine. |
| TextSentencer_T3 |
301-416 |
Sentence |
denotes |
The consequent severe diarrhoea and dehydration are usually fatal unless these sugars are eliminated from the diet. |
| T3 |
301-416 |
Sentence |
denotes |
The consequent severe diarrhoea and dehydration are usually fatal unless these sugars are eliminated from the diet. |
| TextSentencer_T4 |
417-546 |
Sentence |
denotes |
Intestinal biopsies of GGM patients have revealed a specific defect in Na(+)-dependent absorption of glucose in the brush border. |
| T4 |
417-546 |
Sentence |
denotes |
Intestinal biopsies of GGM patients have revealed a specific defect in Na(+)-dependent absorption of glucose in the brush border. |
| TextSentencer_T5 |
547-676 |
Sentence |
denotes |
Normal glucose absorption is mediated by the Na+/glucose cotransporter in the brush border membrane of the intestinal epithelium. |
| T5 |
547-676 |
Sentence |
denotes |
Normal glucose absorption is mediated by the Na+/glucose cotransporter in the brush border membrane of the intestinal epithelium. |
| TextSentencer_T6 |
677-872 |
Sentence |
denotes |
Cellular influx is driven by the transmembrane Na+ electrochemical potential gradient; thereafter the sugar moves to the blood across the basolateral membrane via the facilitated glucose carrier. |
| T6 |
677-872 |
Sentence |
denotes |
Cellular influx is driven by the transmembrane Na+ electrochemical potential gradient; thereafter the sugar moves to the blood across the basolateral membrane via the facilitated glucose carrier. |
| TextSentencer_T7 |
873-1043 |
Sentence |
denotes |
We have previously cloned and sequenced a Na+/glucose cotransporter from normal human ileum and shown that this gene, SGLT1, resides on the distal q arm of chromosome 22. |
| T7 |
873-1043 |
Sentence |
denotes |
We have previously cloned and sequenced a Na+/glucose cotransporter from normal human ileum and shown that this gene, SGLT1, resides on the distal q arm of chromosome 22. |
| TextSentencer_T8 |
1044-1182 |
Sentence |
denotes |
We have now amplified SGLT1 complementary DNA and genomic DNA from members of a family affected with GGM by the polymerase chain reaction. |
| T8 |
1044-1182 |
Sentence |
denotes |
We have now amplified SGLT1 complementary DNA and genomic DNA from members of a family affected with GGM by the polymerase chain reaction. |
| TextSentencer_T9 |
1183-1442 |
Sentence |
denotes |
Sequence analysis of the amplified products has revealed a single missense mutation in SGLT1 which cosegregates with the GGM phenotype and results in a complete loss of Na(+)-dependent glucose transport in Xenopus oocytes injected with this complementary RNA. |
| T9 |
1183-1442 |
Sentence |
denotes |
Sequence analysis of the amplified products has revealed a single missense mutation in SGLT1 which cosegregates with the GGM phenotype and results in a complete loss of Na(+)-dependent glucose transport in Xenopus oocytes injected with this complementary RNA. |