| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-135 |
Sentence |
denotes |
QCM-D studies of human norovirus VLPs binding to glycosphingolipids in supported lipid bilayers reveal strain-specific characteristics. |
| T1 |
0-135 |
Sentence |
denotes |
QCM-D studies of human norovirus VLPs binding to glycosphingolipids in supported lipid bilayers reveal strain-specific characteristics. |
| T1 |
0-135 |
Sentence |
denotes |
QCM-D studies of human norovirus VLPs binding to glycosphingolipids in supported lipid bilayers reveal strain-specific characteristics. |
| TextSentencer_T2 |
136-209 |
Sentence |
denotes |
Susceptibility to norovirus infection has been linked to secretor status. |
| T2 |
136-209 |
Sentence |
denotes |
Susceptibility to norovirus infection has been linked to secretor status. |
| T2 |
136-209 |
Sentence |
denotes |
Susceptibility to norovirus infection has been linked to secretor status. |
| TextSentencer_T3 |
210-445 |
Sentence |
denotes |
Norovirus virus-like particles (VLPs; 0- 20 microg/mL) from the Norwalk (GI.1) and Dijon (GII.4) strains were assayed for binding to H type 1 and Lewis a pentaglycosylceramides, incorporated in laterally fluid supported lipid bilayers. |
| T3 |
210-445 |
Sentence |
denotes |
Norovirus virus-like particles (VLPs; 0- 20 microg/mL) from the Norwalk (GI.1) and Dijon (GII.4) strains were assayed for binding to H type 1 and Lewis a pentaglycosylceramides, incorporated in laterally fluid supported lipid bilayers. |
| T3 |
210-445 |
Sentence |
denotes |
Norovirus virus-like particles (VLPs; 0- 20 microg/mL) from the Norwalk (GI.1) and Dijon (GII.4) strains were assayed for binding to H type 1 and Lewis a pentaglycosylceramides, incorporated in laterally fluid supported lipid bilayers. |
| TextSentencer_T4 |
446-603 |
Sentence |
denotes |
Binding kinetics was monitored in real time in 40 microL stationary reaction chambers, using quartz crystal microbalance with dissipation (QCM-D) monitoring. |
| T4 |
446-603 |
Sentence |
denotes |
Binding kinetics was monitored in real time in 40 microL stationary reaction chambers, using quartz crystal microbalance with dissipation (QCM-D) monitoring. |
| T4 |
446-603 |
Sentence |
denotes |
Binding kinetics was monitored in real time in 40 microL stationary reaction chambers, using quartz crystal microbalance with dissipation (QCM-D) monitoring. |
| TextSentencer_T5 |
604-775 |
Sentence |
denotes |
Both strains displayed binding only to H type 1 and not to Lewis a glycosphingolipids, typical for epithelial cells of susceptible and resistant individuals, respectively. |
| T5 |
604-775 |
Sentence |
denotes |
Both strains displayed binding only to H type 1 and not to Lewis a glycosphingolipids, typical for epithelial cells of susceptible and resistant individuals, respectively. |
| T5 |
604-775 |
Sentence |
denotes |
Both strains displayed binding only to H type 1 and not to Lewis a glycosphingolipids, typical for epithelial cells of susceptible and resistant individuals, respectively. |
| TextSentencer_T6 |
776-891 |
Sentence |
denotes |
This binding specificity was confirmed by VLPs binding to the two glycosphingolipids chromatographed on TLC-plates. |
| T6 |
776-891 |
Sentence |
denotes |
This binding specificity was confirmed by VLPs binding to the two glycosphingolipids chromatographed on TLC-plates. |
| T6 |
776-891 |
Sentence |
denotes |
This binding specificity was confirmed by VLPs binding to the two glycosphingolipids chromatographed on TLC-plates. |
| TextSentencer_T7 |
892-1147 |
Sentence |
denotes |
Experiments using bilayers with mixtures of H type 1 and Lewis a, with the total glycosphingolipid concentration constant at 10 wt%, showed that binding was only dependent on H type 1 concentrations and identical to experiments without additional Lewis a. |
| T7 |
892-1147 |
Sentence |
denotes |
Experiments using bilayers with mixtures of H type 1 and Lewis a, with the total glycosphingolipid concentration constant at 10 wt%, showed that binding was only dependent on H type 1 concentrations and identical to experiments without additional Lewis a. |
| T7 |
892-1147 |
Sentence |
denotes |
Experiments using bilayers with mixtures of H type 1 and Lewis a, with the total glycosphingolipid concentration constant at 10 wt%, showed that binding was only dependent on H type 1 concentrations and identical to experiments without additional Lewis a. |
| TextSentencer_T8 |
1148-1244 |
Sentence |
denotes |
Both strains showed a threshold concentration of H type 1 below which no binding was observable. |
| T8 |
1148-1244 |
Sentence |
denotes |
Both strains showed a threshold concentration of H type 1 below which no binding was observable. |
| T8 |
1148-1244 |
Sentence |
denotes |
Both strains showed a threshold concentration of H type 1 below which no binding was observable. |
| TextSentencer_T9 |
1245-1477 |
Sentence |
denotes |
The threshold was one order of magnitude higher for the Dijon strain (2 wt% versus 0.25 wt%) demonstrating that the interaction with a significantly larger number of glycosphingolipids was needed for the binding of the Dijon strain. |
| T9 |
1245-1477 |
Sentence |
denotes |
The threshold was one order of magnitude higher for the Dijon strain (2 wt% versus 0.25 wt%) demonstrating that the interaction with a significantly larger number of glycosphingolipids was needed for the binding of the Dijon strain. |
| T9 |
1245-1477 |
Sentence |
denotes |
The threshold was one order of magnitude higher for the Dijon strain (2 wt% versus 0.25 wt%) demonstrating that the interaction with a significantly larger number of glycosphingolipids was needed for the binding of the Dijon strain. |
| TextSentencer_T10 |
1478-1656 |
Sentence |
denotes |
The difference in threshold glycosphingolipid concentrations for the two strains suggests a lower affinity for the glycosphingolipid for the Dijon compared to the Norwalk strain. |
| T10 |
1478-1656 |
Sentence |
denotes |
The difference in threshold glycosphingolipid concentrations for the two strains suggests a lower affinity for the glycosphingolipid for the Dijon compared to the Norwalk strain. |
| T10 |
1478-1656 |
Sentence |
denotes |
The difference in threshold glycosphingolipid concentrations for the two strains suggests a lower affinity for the glycosphingolipid for the Dijon compared to the Norwalk strain. |
| TextSentencer_T11 |
1657-1855 |
Sentence |
denotes |
We propose that VLPs initially bind only a few glycosphingolipids but the binding is subsequently strengthened by lateral diffusion of additional glycosphingolipids moving into the interaction area. |
| T11 |
1657-1855 |
Sentence |
denotes |
We propose that VLPs initially bind only a few glycosphingolipids but the binding is subsequently strengthened by lateral diffusion of additional glycosphingolipids moving into the interaction area. |
| T11 |
1657-1855 |
Sentence |
denotes |
We propose that VLPs initially bind only a few glycosphingolipids but the binding is subsequently strengthened by lateral diffusion of additional glycosphingolipids moving into the interaction area. |