| Id |
Subject |
Object |
Predicate |
Lexical cue |
| TextSentencer_T1 |
0-62 |
Sentence |
denotes |
[Retinal vein occlusion and obstructive sleep apnea syndrome]. |
| T1 |
0-62 |
Sentence |
denotes |
[Retinal vein occlusion and obstructive sleep apnea syndrome]. |
| TextSentencer_T2 |
63-124 |
Sentence |
denotes |
Occlusion veineuse rétinienne et syndrome d'apnée du sommeil. |
| T2 |
63-124 |
Sentence |
denotes |
Occlusion veineuse rétinienne et syndrome d'apnée du sommeil. |
| TextSentencer_T3 |
125-133 |
Sentence |
denotes |
PURPOSE: |
| T3 |
125-133 |
Sentence |
denotes |
PURPOSE: |
| TextSentencer_T4 |
134-349 |
Sentence |
denotes |
Obstructive sleep apnea syndrome (OSAS) is a very common disease and has recently been increasingly incriminated in the initiation and progression of numerous cardiovascular, neurologic, and ophthalmologic diseases. |
| T4 |
134-349 |
Sentence |
denotes |
Obstructive sleep apnea syndrome (OSAS) is a very common disease and has recently been increasingly incriminated in the initiation and progression of numerous cardiovascular, neurologic, and ophthalmologic diseases. |
| TextSentencer_T5 |
350-554 |
Sentence |
denotes |
In ophthalmologic practice, it may be related to glaucoma, nonarteritic anterior ischemic optic neuropathy, visual field defects, papilledema, central serous chorioretinopathy, and floppy eyelid syndrome. |
| T5 |
350-554 |
Sentence |
denotes |
In ophthalmologic practice, it may be related to glaucoma, nonarteritic anterior ischemic optic neuropathy, visual field defects, papilledema, central serous chorioretinopathy, and floppy eyelid syndrome. |
| TextSentencer_T6 |
555-648 |
Sentence |
denotes |
Because of personal observations, we investigated its link with retinal vein occlusion (RVO). |
| T6 |
555-648 |
Sentence |
denotes |
Because of personal observations, we investigated its link with retinal vein occlusion (RVO). |
| TextSentencer_T7 |
649-661 |
Sentence |
denotes |
OBSERVATION: |
| T7 |
649-661 |
Sentence |
denotes |
OBSERVATION: |
| TextSentencer_T8 |
662-858 |
Sentence |
denotes |
We report the observations on three patients who presented with retinal vein occlusion and who were investigated positively for OSAS, including patients with no conventional vascular risk factors. |
| T8 |
662-858 |
Sentence |
denotes |
We report the observations on three patients who presented with retinal vein occlusion and who were investigated positively for OSAS, including patients with no conventional vascular risk factors. |
| TextSentencer_T9 |
859-870 |
Sentence |
denotes |
DISCUSSION: |
| T9 |
859-870 |
Sentence |
denotes |
DISCUSSION: |
| TextSentencer_T10 |
871-988 |
Sentence |
denotes |
The local and systemic effects of OSAS could explain, in some patients, the occurrence and/or the aggravation of RVO. |
| T10 |
871-988 |
Sentence |
denotes |
The local and systemic effects of OSAS could explain, in some patients, the occurrence and/or the aggravation of RVO. |
| TextSentencer_T11 |
989-1125 |
Sentence |
denotes |
The first-stage effects of OSAS are nocturnal hypoxemia, hypercapnia, intrathoracic pressure changes, arousals, and sleep fragmentation. |
| T11 |
989-1125 |
Sentence |
denotes |
The first-stage effects of OSAS are nocturnal hypoxemia, hypercapnia, intrathoracic pressure changes, arousals, and sleep fragmentation. |
| TextSentencer_T12 |
1126-1262 |
Sentence |
denotes |
RVO could be a consequence of a slow-down of blood flow circulation secondary to hypoxemia and elevated nocturnal intracranial pressure. |
| T12 |
1126-1262 |
Sentence |
denotes |
RVO could be a consequence of a slow-down of blood flow circulation secondary to hypoxemia and elevated nocturnal intracranial pressure. |
| TextSentencer_T13 |
1263-1338 |
Sentence |
denotes |
The arousals cause an additional acute increase in arterial blood pressure. |
| T13 |
1263-1338 |
Sentence |
denotes |
The arousals cause an additional acute increase in arterial blood pressure. |
| TextSentencer_T14 |
1339-1525 |
Sentence |
denotes |
Ancillary effects include increased platelet aggregability, increased sympathic activation, oxidative stress, vascular endothelial dysfunction, inflammation, and metabolic dysregulation. |
| T14 |
1339-1525 |
Sentence |
denotes |
Ancillary effects include increased platelet aggregability, increased sympathic activation, oxidative stress, vascular endothelial dysfunction, inflammation, and metabolic dysregulation. |
| TextSentencer_T15 |
1526-1537 |
Sentence |
denotes |
CONCLUSION: |
| T15 |
1526-1537 |
Sentence |
denotes |
CONCLUSION: |
| TextSentencer_T16 |
1538-1617 |
Sentence |
denotes |
This short series for the first time reports RVO patients presenting with OSAS. |
| T16 |
1538-1617 |
Sentence |
denotes |
This short series for the first time reports RVO patients presenting with OSAS. |
| TextSentencer_T17 |
1618-1762 |
Sentence |
denotes |
This suggests that OSAS, by acting on retinal microcirculation, could be an additional risk factor for the occurrence or the aggravation of RVO. |
| T17 |
1618-1762 |
Sentence |
denotes |
This suggests that OSAS, by acting on retinal microcirculation, could be an additional risk factor for the occurrence or the aggravation of RVO. |
| TextSentencer_T18 |
1763-1828 |
Sentence |
denotes |
Further studies are needed to confirm this possible relationship. |
| T18 |
1763-1828 |
Sentence |
denotes |
Further studies are needed to confirm this possible relationship. |