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Id Subject Object Predicate Lexical cue
TextSentencer_T1 0-100 Sentence denotes Ganglioside GM3 inhibits VEGF/VEGFR-2-mediated angiogenesis: direct interaction of GM3 with VEGFR-2.
T1 0-100 Sentence denotes Ganglioside GM3 inhibits VEGF/VEGFR-2-mediated angiogenesis: direct interaction of GM3 with VEGFR-2.
T1 0-100 Sentence denotes Ganglioside GM3 inhibits VEGF/VEGFR-2-mediated angiogenesis: direct interaction of GM3 with VEGFR-2.
TextSentencer_T2 101-188 Sentence denotes Angiogenesis is associated with growth, invasion, and metastasis of human solid tumors.
T2 101-188 Sentence denotes Angiogenesis is associated with growth, invasion, and metastasis of human solid tumors.
T2 101-188 Sentence denotes Angiogenesis is associated with growth, invasion, and metastasis of human solid tumors.
TextSentencer_T3 189-488 Sentence denotes Aberrant activation of endothelial cells and induction of microvascular permeability by a vascular endothelial growth factor (VEGF) receptor-2 (VEGFR-2) signaling pathway is observed in pathological angiogenesis including tumor, wound healing, arthritis, psoriasis, diabetic retinopathy, and others.
T3 189-488 Sentence denotes Aberrant activation of endothelial cells and induction of microvascular permeability by a vascular endothelial growth factor (VEGF) receptor-2 (VEGFR-2) signaling pathway is observed in pathological angiogenesis including tumor, wound healing, arthritis, psoriasis, diabetic retinopathy, and others.
T3 189-488 Sentence denotes Aberrant activation of endothelial cells and induction of microvascular permeability by a vascular endothelial growth factor (VEGF) receptor-2 (VEGFR-2) signaling pathway is observed in pathological angiogenesis including tumor, wound healing, arthritis, psoriasis, diabetic retinopathy, and others.
TextSentencer_T4 489-698 Sentence denotes Here, we show that GM3 regulated the activity of various downstream signaling pathways and biological events through the inhibition of VEGF-stimulated VEGFR-2 activation in vascular endothelial cells in vitro.
T4 489-698 Sentence denotes Here, we show that GM3 regulated the activity of various downstream signaling pathways and biological events through the inhibition of VEGF-stimulated VEGFR-2 activation in vascular endothelial cells in vitro.
T4 489-698 Sentence denotes Here, we show that GM3 regulated the activity of various downstream signaling pathways and biological events through the inhibition of VEGF-stimulated VEGFR-2 activation in vascular endothelial cells in vitro.
TextSentencer_T5 699-853 Sentence denotes Furthermore, GM3 strongly blocked VEGF-induced neovascularization in vivo, in models including the chick chorioallantoic membrane and Matrigel plug assay.
T5 699-853 Sentence denotes Furthermore, GM3 strongly blocked VEGF-induced neovascularization in vivo, in models including the chick chorioallantoic membrane and Matrigel plug assay.
T5 699-853 Sentence denotes Furthermore, GM3 strongly blocked VEGF-induced neovascularization in vivo, in models including the chick chorioallantoic membrane and Matrigel plug assay.
TextSentencer_T6 854-1088 Sentence denotes Interestingly, GM3 suppressed VEGF-induced VEGFR-2 activation by blocking its dimerization and also blocked the binding of VEGF to VEGFR-2 through a GM3-specific interaction with the extracellular domain of VEGFR-2, but not with VEGF.
T6 854-1088 Sentence denotes Interestingly, GM3 suppressed VEGF-induced VEGFR-2 activation by blocking its dimerization and also blocked the binding of VEGF to VEGFR-2 through a GM3-specific interaction with the extracellular domain of VEGFR-2, but not with VEGF.
T6 854-1088 Sentence denotes Interestingly, GM3 suppressed VEGF-induced VEGFR-2 activation by blocking its dimerization and also blocked the binding of VEGF to VEGFR-2 through a GM3-specific interaction with the extracellular domain of VEGFR-2, but not with VEGF.
TextSentencer_T7 1089-1165 Sentence denotes Primary tumor growth in mice was inhibited by subcutaneous injection of GM3.
T7 1089-1165 Sentence denotes Primary tumor growth in mice was inhibited by subcutaneous injection of GM3.
T7 1089-1165 Sentence denotes Primary tumor growth in mice was inhibited by subcutaneous injection of GM3.
TextSentencer_T8 1166-1262 Sentence denotes Immunohistochemical analyses showed GM3 inhibition of angiogenesis and tumor cell proliferation.
T8 1166-1262 Sentence denotes Immunohistochemical analyses showed GM3 inhibition of angiogenesis and tumor cell proliferation.
T8 1166-1262 Sentence denotes Immunohistochemical analyses showed GM3 inhibition of angiogenesis and tumor cell proliferation.
TextSentencer_T9 1263-1370 Sentence denotes GM3 also resulted in the suppression of VEGF-stimulated microvessel permeability in mouse skin capillaries.
T9 1263-1370 Sentence denotes GM3 also resulted in the suppression of VEGF-stimulated microvessel permeability in mouse skin capillaries.
T9 1263-1370 Sentence denotes GM3 also resulted in the suppression of VEGF-stimulated microvessel permeability in mouse skin capillaries.
TextSentencer_T10 1371-1541 Sentence denotes These results suggest that GM3 inhibits VEGFR-2-mediated changes in vascular endothelial cell function and angiogenesis, and might be of value in anti-angiogenic therapy.
T10 1371-1541 Sentence denotes These results suggest that GM3 inhibits VEGFR-2-mediated changes in vascular endothelial cell function and angiogenesis, and might be of value in anti-angiogenic therapy.
T10 1371-1541 Sentence denotes These results suggest that GM3 inhibits VEGFR-2-mediated changes in vascular endothelial cell function and angiogenesis, and might be of value in anti-angiogenic therapy.