Id |
Subject |
Object |
Predicate |
Lexical cue |
TextSentencer_T1 |
0-141 |
Sentence |
denotes |
The structure and activities of echinonectin: a developmentally regulated cell adhesion glycoprotein with galactose-specific lectin activity. |
T1 |
0-141 |
Sentence |
denotes |
The structure and activities of echinonectin: a developmentally regulated cell adhesion glycoprotein with galactose-specific lectin activity. |
T1 |
0-141 |
Sentence |
denotes |
The structure and activities of echinonectin: a developmentally regulated cell adhesion glycoprotein with galactose-specific lectin activity. |
TextSentencer_T2 |
142-262 |
Sentence |
denotes |
The extracellular matrix of the sea urchin embryo contains a 230 kD homodimeric glycoprotein known as echinonectin (EN). |
T2 |
142-262 |
Sentence |
denotes |
The extracellular matrix of the sea urchin embryo contains a 230 kD homodimeric glycoprotein known as echinonectin (EN). |
T2 |
142-262 |
Sentence |
denotes |
The extracellular matrix of the sea urchin embryo contains a 230 kD homodimeric glycoprotein known as echinonectin (EN). |
TextSentencer_T3 |
263-348 |
Sentence |
denotes |
EN contains a cell attachment domain as well as a galactose-specific lectin activity. |
T3 |
263-348 |
Sentence |
denotes |
EN contains a cell attachment domain as well as a galactose-specific lectin activity. |
T3 |
263-348 |
Sentence |
denotes |
EN contains a cell attachment domain as well as a galactose-specific lectin activity. |
TextSentencer_T4 |
349-465 |
Sentence |
denotes |
Cell attachment to EN is differentially regulated in the three primary germ layers, endoderm, ectoderm and mesoderm. |
T4 |
349-465 |
Sentence |
denotes |
Cell attachment to EN is differentially regulated in the three primary germ layers, endoderm, ectoderm and mesoderm. |
T4 |
349-465 |
Sentence |
denotes |
Cell attachment to EN is differentially regulated in the three primary germ layers, endoderm, ectoderm and mesoderm. |
TextSentencer_T5 |
466-756 |
Sentence |
denotes |
Prior to gastrulation all embryonic cells adhere equally to EN-coated substrates, but during gastrulation primary mesenchyme cells lose affinity for EN, ectoderm cells increase their binding to the molecule, and cells of the endoderm maintain a similar or slightly lowered level of binding. |
T5 |
466-756 |
Sentence |
denotes |
Prior to gastrulation all embryonic cells adhere equally to EN-coated substrates, but during gastrulation primary mesenchyme cells lose affinity for EN, ectoderm cells increase their binding to the molecule, and cells of the endoderm maintain a similar or slightly lowered level of binding. |
T5 |
466-756 |
Sentence |
denotes |
Prior to gastrulation all embryonic cells adhere equally to EN-coated substrates, but during gastrulation primary mesenchyme cells lose affinity for EN, ectoderm cells increase their binding to the molecule, and cells of the endoderm maintain a similar or slightly lowered level of binding. |
TextSentencer_T6 |
757-887 |
Sentence |
denotes |
The mechanisms governing these adhesive changes and the specific functions they serve in development are not currently understood. |
T6 |
757-887 |
Sentence |
denotes |
The mechanisms governing these adhesive changes and the specific functions they serve in development are not currently understood. |
T6 |
757-887 |
Sentence |
denotes |
The mechanisms governing these adhesive changes and the specific functions they serve in development are not currently understood. |
TextSentencer_T7 |
888-1117 |
Sentence |
denotes |
They are timed to coincide with distinct morphogenetic events such as primary mesenchyme cell ingression and archenteron formation, suggesting that regulated adhesion to EN plays at least a permissive role in early morphogenesis. |
T7 |
888-1117 |
Sentence |
denotes |
They are timed to coincide with distinct morphogenetic events such as primary mesenchyme cell ingression and archenteron formation, suggesting that regulated adhesion to EN plays at least a permissive role in early morphogenesis. |
T7 |
888-1117 |
Sentence |
denotes |
They are timed to coincide with distinct morphogenetic events such as primary mesenchyme cell ingression and archenteron formation, suggesting that regulated adhesion to EN plays at least a permissive role in early morphogenesis. |