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Id Subject Object Predicate Lexical cue
TextSentencer_T1 0-117 Sentence denotes Hypoglycosylation with increased fucosylation and branching of serum transferrin N-glycans in untreated galactosemia.
T1 0-117 Sentence denotes Hypoglycosylation with increased fucosylation and branching of serum transferrin N-glycans in untreated galactosemia.
T1 0-117 Sentence denotes Hypoglycosylation with increased fucosylation and branching of serum transferrin N-glycans in untreated galactosemia.
TextSentencer_T2 118-378 Sentence denotes Untreated classic galactosemia (galactose-1-phosphate uridyltransferase [GALT] deficiency) is known as a secondary congenital disorders of glycosylation (CDG) characterized by galactose deficiency of glycoproteins and glycolipids (processing defect or CDG-II).
T2 118-378 Sentence denotes Untreated classic galactosemia (galactose-1-phosphate uridyltransferase [GALT] deficiency) is known as a secondary congenital disorders of glycosylation (CDG) characterized by galactose deficiency of glycoproteins and glycolipids (processing defect or CDG-II).
T2 118-378 Sentence denotes Untreated classic galactosemia (galactose-1-phosphate uridyltransferase [GALT] deficiency) is known as a secondary congenital disorders of glycosylation (CDG) characterized by galactose deficiency of glycoproteins and glycolipids (processing defect or CDG-II).
TextSentencer_T3 379-447 Sentence denotes The mechanism of this undergalactosylation has not been established.
T3 379-447 Sentence denotes The mechanism of this undergalactosylation has not been established.
T3 379-447 Sentence denotes The mechanism of this undergalactosylation has not been established.
TextSentencer_T4 448-677 Sentence denotes Here we show that in untreated galactosemia, there is also a partial deficiency of whole glycans of serum transferrin associated with increased fucosylation and branching as seen in genetic glycosylation assembly defects (CDG-I).
T4 448-677 Sentence denotes Here we show that in untreated galactosemia, there is also a partial deficiency of whole glycans of serum transferrin associated with increased fucosylation and branching as seen in genetic glycosylation assembly defects (CDG-I).
T4 448-677 Sentence denotes Here we show that in untreated galactosemia, there is also a partial deficiency of whole glycans of serum transferrin associated with increased fucosylation and branching as seen in genetic glycosylation assembly defects (CDG-I).
TextSentencer_T5 678-798 Sentence denotes Thus galactosemia seems to be a secondary "dual" CDG causing a processing as well as an assembly N-glycosylation defect.
T5 678-798 Sentence denotes Thus galactosemia seems to be a secondary "dual" CDG causing a processing as well as an assembly N-glycosylation defect.
T5 678-798 Sentence denotes Thus galactosemia seems to be a secondary "dual" CDG causing a processing as well as an assembly N-glycosylation defect.
TextSentencer_T6 799-920 Sentence denotes We also demonstrated that in galactosemia patients, transferrin N-glycan biosynthesis is restored upon dietary treatment.
T6 799-920 Sentence denotes We also demonstrated that in galactosemia patients, transferrin N-glycan biosynthesis is restored upon dietary treatment.
T6 799-920 Sentence denotes We also demonstrated that in galactosemia patients, transferrin N-glycan biosynthesis is restored upon dietary treatment.