Id |
Subject |
Object |
Predicate |
Lexical cue |
TextSentencer_T1 |
0-101 |
Sentence |
denotes |
Testis-specific sulfoglycolipid, seminolipid, is essential for germ cell function in spermatogenesis. |
T1 |
0-101 |
Sentence |
denotes |
Testis-specific sulfoglycolipid, seminolipid, is essential for germ cell function in spermatogenesis. |
T1 |
0-101 |
Sentence |
denotes |
Testis-specific sulfoglycolipid, seminolipid, is essential for germ cell function in spermatogenesis. |
TextSentencer_T2 |
102-215 |
Sentence |
denotes |
More than 90% of the glycolipid in mammalian testis consists of a unique sulfated glyceroglycolipid, seminolipid. |
T2 |
102-215 |
Sentence |
denotes |
More than 90% of the glycolipid in mammalian testis consists of a unique sulfated glyceroglycolipid, seminolipid. |
T2 |
102-215 |
Sentence |
denotes |
More than 90% of the glycolipid in mammalian testis consists of a unique sulfated glyceroglycolipid, seminolipid. |
TextSentencer_T3 |
216-343 |
Sentence |
denotes |
The sulfation of the molecule is catalyzed by a Golgi membrane-associated sulfotransferase, cerebroside sulfotransferase (CST). |
T3 |
216-343 |
Sentence |
denotes |
The sulfation of the molecule is catalyzed by a Golgi membrane-associated sulfotransferase, cerebroside sulfotransferase (CST). |
T3 |
216-343 |
Sentence |
denotes |
The sulfation of the molecule is catalyzed by a Golgi membrane-associated sulfotransferase, cerebroside sulfotransferase (CST). |
TextSentencer_T4 |
344-488 |
Sentence |
denotes |
Disruption of the Cst gene in mice results in male infertility due to the arrest of spermatogenesis prior to the metaphase of the first meiosis. |
T4 |
344-488 |
Sentence |
denotes |
Disruption of the Cst gene in mice results in male infertility due to the arrest of spermatogenesis prior to the metaphase of the first meiosis. |
T4 |
344-488 |
Sentence |
denotes |
Disruption of the Cst gene in mice results in male infertility due to the arrest of spermatogenesis prior to the metaphase of the first meiosis. |
TextSentencer_T5 |
489-624 |
Sentence |
denotes |
However, the issue of which side of the cell function-germ cells or Sertoli cells-is deteriorated in this mutant mouse remains unknown. |
T5 |
489-624 |
Sentence |
denotes |
However, the issue of which side of the cell function-germ cells or Sertoli cells-is deteriorated in this mutant mouse remains unknown. |
T5 |
489-624 |
Sentence |
denotes |
However, the issue of which side of the cell function-germ cells or Sertoli cells-is deteriorated in this mutant mouse remains unknown. |
TextSentencer_T6 |
625-866 |
Sentence |
denotes |
Our findings show that the defect is in the germ cell side, as evidenced by a transplantation analysis, in which wild-type spermatogonia expressing the green fluorescent protein were injected into the seminiferous tubules of CST-null testis. |
T6 |
625-866 |
Sentence |
denotes |
Our findings show that the defect is in the germ cell side, as evidenced by a transplantation analysis, in which wild-type spermatogonia expressing the green fluorescent protein were injected into the seminiferous tubules of CST-null testis. |
T6 |
625-866 |
Sentence |
denotes |
Our findings show that the defect is in the germ cell side, as evidenced by a transplantation analysis, in which wild-type spermatogonia expressing the green fluorescent protein were injected into the seminiferous tubules of CST-null testis. |
TextSentencer_T7 |
867-986 |
Sentence |
denotes |
The transplanted GFP-positive cells generated colonies and spermatogenesis proceeded over meiosis in the mutant testis. |
T7 |
867-986 |
Sentence |
denotes |
The transplanted GFP-positive cells generated colonies and spermatogenesis proceeded over meiosis in the mutant testis. |
T7 |
867-986 |
Sentence |
denotes |
The transplanted GFP-positive cells generated colonies and spermatogenesis proceeded over meiosis in the mutant testis. |
TextSentencer_T8 |
987-1389 |
Sentence |
denotes |
The findings also clearly show that the seminolipid is expressed on the plasma membranes of spermatogonia, spermatocytes, spermatids, and spermatozoa, as evidenced by the immunostaining of wild-type testes using an anti-sulfogalactolipid antibody, Sulph-1 in comparison with CST-null testes as a negative control, and that seminolipid appears as early as day 8 of age, when Type B spermatogonia emerge. |
T8 |
987-1389 |
Sentence |
denotes |
The findings also clearly show that the seminolipid is expressed on the plasma membranes of spermatogonia, spermatocytes, spermatids, and spermatozoa, as evidenced by the immunostaining of wild-type testes using an anti-sulfogalactolipid antibody, Sulph-1 in comparison with CST-null testes as a negative control, and that seminolipid appears as early as day 8 of age, when Type B spermatogonia emerge. |
T8 |
987-1389 |
Sentence |
denotes |
The findings also clearly show that the seminolipid is expressed on the plasma membranes of spermatogonia, spermatocytes, spermatids, and spermatozoa, as evidenced by the immunostaining of wild-type testes using an anti-sulfogalactolipid antibody, Sulph-1 in comparison with CST-null testes as a negative control, and that seminolipid appears as early as day 8 of age, when Type B spermatogonia emerge. |