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PubMed:15046742 JSONTXT 7 Projects

Annnotations TAB TSV DIC JSON TextAE Lectin_function IAV-Glycan

Id Subject Object Predicate Lexical cue
TextSentencer_T1 0-78 Sentence denotes Disregulated glyceroneogenesis: PCK1 as a candidate diabetes and obesity gene.
T1 0-78 Sentence denotes Disregulated glyceroneogenesis: PCK1 as a candidate diabetes and obesity gene.
TextSentencer_T2 79-152 Sentence denotes Genetics and diet interact to cause type 2 diabetes mellitus and obesity.
T2 79-152 Sentence denotes Genetics and diet interact to cause type 2 diabetes mellitus and obesity.
TextSentencer_T3 153-240 Sentence denotes PCK1 has been implicated as one of many genes associated with type 2 diabetes mellitus.
T3 153-240 Sentence denotes PCK1 has been implicated as one of many genes associated with type 2 diabetes mellitus.
TextSentencer_T4 241-358 Sentence denotes The common assumption is that mutations in PCK1 lead to excessive glucose production through hepatic gluconeogenesis.
T4 241-358 Sentence denotes The common assumption is that mutations in PCK1 lead to excessive glucose production through hepatic gluconeogenesis.
TextSentencer_T5 359-500 Sentence denotes However, there is an alternative explanation, wherein mutations at the PCK1 locus could selectively affect PCK1 expression in adipose tissue.
T5 359-500 Sentence denotes However, there is an alternative explanation, wherein mutations at the PCK1 locus could selectively affect PCK1 expression in adipose tissue.
TextSentencer_T6 501-607 Sentence denotes The result would be changes in glyceroneogenesis that would affect the storage and release of fatty acids.
T6 501-607 Sentence denotes The result would be changes in glyceroneogenesis that would affect the storage and release of fatty acids.
TextSentencer_T7 608-758 Sentence denotes Here, we present the novel hypothesis that a variety of phenotypes could arise from mutations of the various tissue-specific control elements of PCK1.
T7 608-758 Sentence denotes Here, we present the novel hypothesis that a variety of phenotypes could arise from mutations of the various tissue-specific control elements of PCK1.
TextSentencer_T8 759-903 Sentence denotes We also suggest specific quantitative metabolic traits that would accompany mutations that selectively affect PCK1 expression in adipose tissue.
T8 759-903 Sentence denotes We also suggest specific quantitative metabolic traits that would accompany mutations that selectively affect PCK1 expression in adipose tissue.