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PubMed:10910970 JSONTXT 53 Projects

Annnotations TAB TSV DIC JSON TextAE Lectin_function IAV-Glycan

Id Subject Object Predicate Lexical cue
TextSentencer_T1 0-48 Sentence denotes Influenza virus infection of desialylated cells.
T1 0-48 Sentence denotes Influenza virus infection of desialylated cells.
T1 0-48 Sentence denotes Influenza virus infection of desialylated cells.
TextSentencer_T2 49-130 Sentence denotes Sialic acid has long been considered to be the sole receptor for influenza virus.
T2 49-130 Sentence denotes Sialic acid has long been considered to be the sole receptor for influenza virus.
T2 49-130 Sentence denotes Sialic acid has long been considered to be the sole receptor for influenza virus.
TextSentencer_T3 131-344 Sentence denotes The viral hemagglutinin (HA) is known to bind cell surface sialic acid, and sialic acids on viral glyco-proteins are cleaved by the viral neuraminidase (NA) to promote efficient release of progeny virus particles.
T3 131-344 Sentence denotes The viral hemagglutinin (HA) is known to bind cell surface sialic acid, and sialic acids on viral glyco-proteins are cleaved by the viral neuraminidase (NA) to promote efficient release of progeny virus particles.
T3 131-344 Sentence denotes The viral hemagglutinin (HA) is known to bind cell surface sialic acid, and sialic acids on viral glyco-proteins are cleaved by the viral neuraminidase (NA) to promote efficient release of progeny virus particles.
TextSentencer_T4 345-488 Sentence denotes However, NWS-Mvi, a mutant virus completely lacking NA, grows well in MDCK cells continuously treated with exogenous neuraminidase (sialidase).
T4 345-488 Sentence denotes However, NWS-Mvi, a mutant virus completely lacking NA, grows well in MDCK cells continuously treated with exogenous neuraminidase (sialidase).
T4 345-488 Sentence denotes However, NWS-Mvi, a mutant virus completely lacking NA, grows well in MDCK cells continuously treated with exogenous neuraminidase (sialidase).
TextSentencer_T5 489-670 Sentence denotes Exogenous sialidase quantitatively releases all sialic acids from purified glycoproteins and glycolipids of MDCK cells and efficiently removes surface sialic acid from intact cells.
T5 489-670 Sentence denotes Exogenous sialidase quantitatively releases all sialic acids from purified glycoproteins and glycolipids of MDCK cells and efficiently removes surface sialic acid from intact cells.
T5 489-670 Sentence denotes Exogenous sialidase quantitatively releases all sialic acids from purified glycoproteins and glycolipids of MDCK cells and efficiently removes surface sialic acid from intact cells.
TextSentencer_T6 671-821 Sentence denotes Binding of NWS-Mvi and parent influenza viruses to MDCK cells is indistinguishable, and is only partially reduced by sialidase treatment of the cells.
T6 671-821 Sentence denotes Binding of NWS-Mvi and parent influenza viruses to MDCK cells is indistinguishable, and is only partially reduced by sialidase treatment of the cells.
T6 671-821 Sentence denotes Binding of NWS-Mvi and parent influenza viruses to MDCK cells is indistinguishable, and is only partially reduced by sialidase treatment of the cells.
TextSentencer_T7 822-922 Sentence denotes Both mutant and wild-type viruses enter enzymatically desialylated cells and initiate transcription.
T7 822-922 Sentence denotes Both mutant and wild-type viruses enter enzymatically desialylated cells and initiate transcription.
T7 822-922 Sentence denotes Both mutant and wild-type viruses enter enzymatically desialylated cells and initiate transcription.
TextSentencer_T8 923-1113 Sentence denotes The ability of influenza A reassortant viruses to infect desialylated cells is shared by recent H3N2 clinical isolates, suggesting that this may be a general property of influenza A viruses.
T8 923-1113 Sentence denotes The ability of influenza A reassortant viruses to infect desialylated cells is shared by recent H3N2 clinical isolates, suggesting that this may be a general property of influenza A viruses.
T8 923-1113 Sentence denotes The ability of influenza A reassortant viruses to infect desialylated cells is shared by recent H3N2 clinical isolates, suggesting that this may be a general property of influenza A viruses.
TextSentencer_T9 1114-1250 Sentence denotes We propose that influenza virus infection can result from sialic acid-independent receptors, either directly or in a multistage process.
T9 1114-1250 Sentence denotes We propose that influenza virus infection can result from sialic acid-independent receptors, either directly or in a multistage process.
T9 1114-1250 Sentence denotes We propose that influenza virus infection can result from sialic acid-independent receptors, either directly or in a multistage process.
TextSentencer_T10 1251-1402 Sentence denotes When sialic acid is present, it may act to enhance virus binding to the cell surface to increase interaction with secondary receptors to mediate entry.
T10 1251-1402 Sentence denotes When sialic acid is present, it may act to enhance virus binding to the cell surface to increase interaction with secondary receptors to mediate entry.
T10 1251-1402 Sentence denotes When sialic acid is present, it may act to enhance virus binding to the cell surface to increase interaction with secondary receptors to mediate entry.
TextSentencer_T11 1403-1501 Sentence denotes Understanding virus entry will be critical to further efforts in infection control and prevention.
T11 1403-1501 Sentence denotes Understanding virus entry will be critical to further efforts in infection control and prevention.
T11 1403-1501 Sentence denotes Understanding virus entry will be critical to further efforts in infection control and prevention.