| Id |
Subject |
Object |
Predicate |
Lexical cue |
| T6210 |
0-151 |
Sentence |
denotes |
Toll-like receptors (TLRs) are a family of signal transduction molecules and play a critical role in the induction of innate and adaptive immunity [4]. |
| T144 |
0-151 |
Sentence |
denotes |
Toll-like receptors (TLRs) are a family of signal transduction molecules and play a critical role in the induction of innate and adaptive immunity [4]. |
| T6211 |
152-233 |
Sentence |
denotes |
TLR-mediated signaling pathways mainly stimulate the activation of NFκB [12, 13]. |
| T145 |
152-233 |
Sentence |
denotes |
TLR-mediated signaling pathways mainly stimulate the activation of NFκB [12, 13]. |
| T6212 |
234-443 |
Sentence |
denotes |
TLR4, the first mammalian TLR recognized, has been reported to be involved in several central nervous (CNS) system diseases, such as inflammatory or autoimmune CNS diseases and cerebral ischemic injury [7, 8]. |
| T146 |
234-443 |
Sentence |
denotes |
TLR4, the first mammalian TLR recognized, has been reported to be involved in several central nervous (CNS) system diseases, such as inflammatory or autoimmune CNS diseases and cerebral ischemic injury [7, 8]. |
| T6213 |
444-766 |
Sentence |
denotes |
Although previous reports indicated that the activation of NFκB was attributed to the oxidative stress or glutamate receptor activation induced by red blood cells and/or plasma constituents [17], very few studies have been directed to investigate the role and the relationship between TLR4-mediated NFκB signaling and ICH. |
| T147 |
444-766 |
Sentence |
denotes |
Although previous reports indicated that the activation of NFκB was attributed to the oxidative stress or glutamate receptor activation induced by red blood cells and/or plasma constituents [17], very few studies have been directed to investigate the role and the relationship between TLR4-mediated NFκB signaling and ICH. |
