Id |
Subject |
Object |
Predicate |
Lexical cue |
T6204 |
0-269 |
Sentence |
denotes |
Transcription factor nuclear factor-kappa-B (NFκB) is an important nuclear transcription factor, which initiates transcription of genes associated with immune responses and inflammation [12, 13] and also plays a key role in regulating inflammation in brain pathologies. |
T138 |
0-269 |
Sentence |
denotes |
Transcription factor nuclear factor-kappa-B (NFκB) is an important nuclear transcription factor, which initiates transcription of genes associated with immune responses and inflammation [12, 13] and also plays a key role in regulating inflammation in brain pathologies. |
T6205 |
270-356 |
Sentence |
denotes |
It has been reported that NFκB increased in the region of perihematoma after ICH [14]. |
T139 |
270-356 |
Sentence |
denotes |
It has been reported that NFκB increased in the region of perihematoma after ICH [14]. |
T6206 |
357-518 |
Sentence |
denotes |
NFκB activation occurs within minutes and persists for at least a week in response to ICH, which is associated with the expression of selected target genes [15]. |
T140 |
357-518 |
Sentence |
denotes |
NFκB activation occurs within minutes and persists for at least a week in response to ICH, which is associated with the expression of selected target genes [15]. |
T6207 |
519-649 |
Sentence |
denotes |
Activation of NFκB initiates inflammatory responses and contributes to the pathobiology of perilesional cell death after ICH [16]. |
T141 |
519-649 |
Sentence |
denotes |
Activation of NFκB initiates inflammatory responses and contributes to the pathobiology of perilesional cell death after ICH [16]. |
T6208 |
650-837 |
Sentence |
denotes |
Inhibition of NFκB activity has been shown to have a therapeutic effect on experimental ICH, such as reduce inflammation, behavioral dysfunction, and neuronal damage produced by ICH [17]. |
T142 |
650-837 |
Sentence |
denotes |
Inhibition of NFκB activity has been shown to have a therapeutic effect on experimental ICH, such as reduce inflammation, behavioral dysfunction, and neuronal damage produced by ICH [17]. |
T6209 |
838-971 |
Sentence |
denotes |
The knowledge about how NFκB is activated in the brain subjected to ICH is critical for seeking a new neuroprotective method for ICH. |
T143 |
838-971 |
Sentence |
denotes |
The knowledge about how NFκB is activated in the brain subjected to ICH is critical for seeking a new neuroprotective method for ICH. |