| Id |
Subject |
Object |
Predicate |
Lexical cue |
| T64 |
0-65 |
Sentence |
denotes |
Traditionally, G6PD studies have been focused on human red cells. |
| T65 |
66-205 |
Sentence |
denotes |
G6PD in nucleated cells regulates cellular processes, including cell proliferation, cell death, autophagy, inflammation, and tumorigenesis. |
| T66 |
206-313 |
Sentence |
denotes |
G6PD deficiency reduces replicative potential in human fibroblasts, leading to early-onset senescence [27]. |
| T67 |
314-430 |
Sentence |
denotes |
Such premature senescence is most likely due to elevated oxidative stress rather than increased telomere shortening. |
| T68 |
431-608 |
Sentence |
denotes |
Approaches using biochemical inhibitors or RNAi knockdown against G6PD in several cell lines demonstrate that decreased G6PD activity is associated with growth retardation [28]. |
| T69 |
609-693 |
Sentence |
denotes |
The most common form of cell death caused by G6PD activity suppression is apoptosis. |
