LitCovid-sentences  

PMC:7799377 / 41567-44489 JSONTXT 3 Projects

Annnotations TAB TSV DIC JSON TextAE

Id Subject Object Predicate Lexical cue
T275 0-10 Sentence denotes Discussion
T276 11-247 Sentence denotes As scientists and clinicians concentrate on the complexities of the SARS-CoV-2 virus and better treatment strategies for COVID-19, it is possible that neurological manifestations of COVID-19 are being overlooked or misinterpreted [117].
T277 248-524 Sentence denotes In general, neurological symptoms can be under-diagnosed or even entirely overlooked as neurological manifestations, such as delirium in the critically ill COVID-19 patients or infected outpatients who do not consider a loss of the sense of smell or taste related to COVID-19.
T278 525-661 Sentence denotes As such, clinicians on the front lines of COVID-19 care should be cognizant of possible neurological manifestations of this novel virus.
T279 662-847 Sentence denotes Patients with suspected or diagnosed COVID-19 should be asked about the loss of smell and taste and educated that this is an important symptom to bring to the attention of the provider.
T280 848-954 Sentence denotes Neurological symptoms of COVID-19 have to date mainly be described within the trajectory of the infection.
T281 955-1167 Sentence denotes However, it is plausible (although far from established) that neurological sequelae from COVID-19 may emerge after the patient has recovered from the primary infection and persist for long periods after recovery.
T282 1168-1332 Sentence denotes For example, it is not known if an elderly patient who recovers from a severe case of COVID-19 with cognitive dysfunction will suffer persistent cognitive deficits.
T283 1333-1593 Sentence denotes The long-term burden on both caregivers and the healthcare system that might be posed by COVID-19 survivors with neurological or cognitive impairment may turn out to be very important, although it is one that is rarely discussed, even hypothetically [123,124].
T284 1594-1738 Sentence denotes There is evidence of neurological complications with SARS and MERS, and a growing body of evidence for neurological complications with COVID-19.
T285 1739-2017 Sentence denotes The etiology of these neurological symptoms is less clear; they may be directly caused by the viral infection or they could be due to other conditions, such as sepsis, coagulation disorders, cytokine release, and vasculitis, all of which have been reported in COVID-19 patients.
T286 2018-2162 Sentence denotes Much more needs to be learned, but clinicians must be aware and prepared for the possibility and potentiality of COVID-19 neurological symptoms.
T287 2163-2226 Sentence denotes The evidence that the SARS-CoV-2 can enter the CNS is alarming.
T288 2227-2385 Sentence denotes While autopsy studies show definitive proof that the earlier SARS-CoV was found in brain tissue, viral levels were lower in the brain than in the lungs [125].
T289 2386-2492 Sentence denotes The route by which the SARS-CoV-2 virus enters the CNS and its effects on the CNS remain to be elucidated.
T290 2493-2585 Sentence denotes It is important to learn more about chronic neurological complications related to COVID-019.
T291 2586-2768 Sentence denotes The blood-brain barrier, which may protect patients from invading pathogens, is a two-way street and could in theory at least prevent viruses from being expelled from the brain [50].
T292 2769-2922 Sentence denotes Further study is needed, particularly as the population of COVID-recovered patients grows and may have to manage long-term consequences of the infection.