| Id |
Subject |
Object |
Predicate |
Lexical cue |
| T59 |
0-119 |
Sentence |
denotes |
RvE2 is produced by reduction of 18R HEPE products by 5-LOX to 5S-hydroperoxy, 18-hydroxy-EPE in whole blood (Oh et al. |
| T60 |
120-126 |
Sentence |
denotes |
2012). |
| T61 |
127-240 |
Sentence |
denotes |
Unlike RvE1, RvE2 and RvE3 are biosynthesized from 18-HEPE via the 12/15-LOX pathway in eosinophils (Isobe et al. |
| T62 |
241-248 |
Sentence |
denotes |
2012b). |
| T63 |
249-381 |
Sentence |
denotes |
Endogenous RvE1 has been shown to accumulate for between 48 and 72 hours, which is a delayed time point of inflammation (Hong et al. |
| T64 |
382-388 |
Sentence |
denotes |
2008). |
| T65 |
389-556 |
Sentence |
denotes |
RvE2 appeared at the time point corresponding to initial PMN infiltration in rat peritoneal exudate stimulated by zymosan A and decreased within 24 hours (Isobe et al. |
| T66 |
557-564 |
Sentence |
denotes |
2012a). |
| T67 |
565-654 |
Sentence |
denotes |
18S-RvE1 is produced by 5-LOX and LTA4 hydrolase using 18S-HEPE as a substrate (Oh et al. |
| T68 |
655-661 |
Sentence |
denotes |
2011). |
