Id |
Subject |
Object |
Predicate |
Lexical cue |
T330 |
0-146 |
Sentence |
denotes |
Hepatitis C virus (HCV) is a positive-sense single-stranded RNA virus of the family Flaviviridae with a small (55–65 nm size) envelope (Lee et al. |
T331 |
147-153 |
Sentence |
denotes |
2017). |
T332 |
154-308 |
Sentence |
denotes |
The HCV is the cause of hepatitis C and some cancers such as liver cancer (hepatocellular carcinoma, abbreviated HCC) and lymphoma in humans (Ferri et al. |
T333 |
309-337 |
Sentence |
denotes |
2015; Rusyn and Lemon 2014). |
T334 |
338-418 |
Sentence |
denotes |
To date, there appears to be no report on the effectiveness of SPMs against HCV. |
T335 |
419-566 |
Sentence |
denotes |
However, HCV peptide (C5A), an amphiphilic α-helix peptide of HCV, is an activator of the N-formyl peptide receptor in human phagocytes (Lin et al. |
T336 |
567-573 |
Sentence |
denotes |
2011). |
T337 |
574-664 |
Sentence |
denotes |
This suggests the possibility of interaction between RvD1 and LXA4, FPR families, and HCV. |
T338 |
665-787 |
Sentence |
denotes |
Vitamin D metabolites inhibit HCV and upregulate GPR37 gene expression, which induces cellular autophagy (Gutierrez et al. |
T339 |
788-794 |
Sentence |
denotes |
2014). |
T340 |
795-926 |
Sentence |
denotes |
PD1 was recently proposed as a new ligand for GPR37, and some studies suggest a possible relationship between PD1 and HCV (Fig. 4). |
T341 |
927-1077 |
Sentence |
denotes |
SAA also has antiviral effects against HCV, however, it induces chronic inflammation through FPR2/ALX, causing liver damage (Abouelasrar Salama et al. |
T342 |
1078-1084 |
Sentence |
denotes |
2019). |
T343 |
1085-1250 |
Sentence |
denotes |
Although research has not been conducted yet, RvD1 and LXA4, which inhibit the action of SAA, are likely to suppress liver damage caused by SAA during HCV infection. |