Id |
Subject |
Object |
Predicate |
Lexical cue |
T288 |
0-20 |
Sentence |
denotes |
Herpes simplex virus |
T289 |
21-270 |
Sentence |
denotes |
Herpesviruses 1 and 2 (HSV-1 and HSV-2), with the taxonomic names human alphaherpesvirus 1 and human alphaherpesvirus 2, are the most common causes of human viral infections among the members of the human Herpesviridae family (Chayavichitsilp et al. |
T290 |
271-277 |
Sentence |
denotes |
2009). |
T291 |
278-617 |
Sentence |
denotes |
Herpes simplex virus (HSV) ocular infection represents another example in which local control of the virus stems from a robust inflammatory response with long-term consequences of chronic inflammation, including the possibility of final blindness due to interstitial keratitis that persists even after the viral infection has been cleared. |
T292 |
618-912 |
Sentence |
denotes |
In animals with HSV, topical administration of RvE1 reduced the influx of CD4+ T cells (both TH1 cells and TH17 cells) and neutrophils, decreased production of inflammatory cytokines including IFNγ and IL-6, and increased levels of the anti-inflammatory cytokine IL-10 (Fig. 4) (Rajasagi et al. |
T293 |
913-919 |
Sentence |
denotes |
2011). |
T294 |
920-974 |
Sentence |
denotes |
Overall, RvE1 significantly reduced stromal keratitis. |
T295 |
975-1043 |
Sentence |
denotes |
Similar results were demonstrated with PD1 (Fig. 4) (Rajasagi et al. |
T296 |
1044-1050 |
Sentence |
denotes |
2013). |
T297 |
1051-1351 |
Sentence |
denotes |
Besides, AT-RvD1 treatment significantly reduced the degree of corneal angiogenesis and the severity of stromal keratitis lesions, and AT-RvD1 treated mice had fewer Th1 and Th17 cells in the infected cornea, as well as the reduced number of inflammatory cells, including neutrophils (Rajasagi et al. |
T298 |
1352-1358 |
Sentence |
denotes |
2017). |