| Id |
Subject |
Object |
Predicate |
Lexical cue |
| T458 |
0-91 |
Sentence |
denotes |
Pulmonary ACE2 is potentially a candidate target in CoV-involved inflammatory pathogenesis. |
| T459 |
92-174 |
Sentence |
denotes |
If ACE inhibitors and Ang II-AT1 blockers are dosed, ACE2 expression is increased. |
| T460 |
175-279 |
Sentence |
denotes |
However, currently we have no conclusive evidence that the inhibitors help SARS-CoV or SARS-CoV-2 entry. |
| T461 |
280-331 |
Sentence |
denotes |
Rather, SARS-CoV infection reduces ACE2 expression. |
| T462 |
332-401 |
Sentence |
denotes |
Therefore, SARS-CoV-2 host tropism is not related to ACE2 expression. |
| T463 |
402-476 |
Sentence |
denotes |
ACE2 levels and ANG II/ANG 1–7 levels regulate the pathogenic progression. |
| T464 |
477-601 |
Sentence |
denotes |
ACE2 expression is upregulated by gene polymorphisms and ACE inhibitors or Angiotensin II receptor blockers such as sartans. |
