PubMed:26102294 / 1183-1465 JSONTXT 13 Projects

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Id Subject Object Predicate Lexical cue
T8 0-282 Sentence denotes Instead, ETO-induced OCT4A was concomitant with activation of AMPK, a key component of metabolic stress and autophagy regulation. p16ink4a, the inducer of terminal senescence, underwent autophagic sequestration in the cytoplasm of ETO-treated cells, allowing alternative cell fates.