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PubMed:3925797 JSONTXT 4 Projects

Annnotations TAB TSV DIC JSON TextAE Lectin_function IAV-Glycan

Id Subject Object Predicate Lexical cue
T1 0-104 Sentence denotes Prostaglandin involvement in hypersensitivity of ischemic hearts to arrhythmogenic influence of ouabain.
T1 0-104 Sentence denotes Prostaglandin involvement in hypersensitivity of ischemic hearts to arrhythmogenic influence of ouabain.
T2 105-198 Sentence denotes Arrhythmias were produced by ouabain (2.7 X 10(-7) M) in isolated perfused guinea pig hearts.
T2 105-198 Sentence denotes Arrhythmias were produced by ouabain (2.7 X 10(-7) M) in isolated perfused guinea pig hearts.
T3 199-313 Sentence denotes In control hearts the average time to onset of arrhythmias following exposure to ouabain was approximately 35 min.
T3 199-313 Sentence denotes In control hearts the average time to onset of arrhythmias following exposure to ouabain was approximately 35 min.
T4 314-435 Sentence denotes Ligation of the left anterior descending coronary artery significantly shortened the time to arrhythmias to about 15 min.
T4 314-435 Sentence denotes Ligation of the left anterior descending coronary artery significantly shortened the time to arrhythmias to about 15 min.
T5 436-594 Sentence denotes This effect of ligation was attenuated significantly by pretreating the hearts with acetylsalicylic acid (ASA) and other nonsteroidal anti-inflammatory drugs.
T5 436-594 Sentence denotes This effect of ligation was attenuated significantly by pretreating the hearts with acetylsalicylic acid (ASA) and other nonsteroidal anti-inflammatory drugs.
T6 595-723 Sentence denotes In contrast, the protective influence of ASA was reversed by either prostaglandin (PG) E2, PGF2 alpha, or PGI2 (2.8 X 10(-9) M).
T6 595-723 Sentence denotes In contrast, the protective influence of ASA was reversed by either prostaglandin (PG) E2, PGF2 alpha, or PGI2 (2.8 X 10(-9) M).
T7 724-863 Sentence denotes When PGE2 or PGI2 were present in the absence of ASA, the time to arrhythmias following ouabain administration was significantly shortened.
T7 724-863 Sentence denotes When PGE2 or PGI2 were present in the absence of ASA, the time to arrhythmias following ouabain administration was significantly shortened.
T8 864-910 Sentence denotes PGF2 alpha had no effect in these experiments.
T8 864-910 Sentence denotes PGF2 alpha had no effect in these experiments.
T9 911-989 Sentence denotes The sensitivity was enhanced significantly by treatment with arachidonic acid.
T9 911-989 Sentence denotes The sensitivity was enhanced significantly by treatment with arachidonic acid.
T10 990-1052 Sentence denotes This effect was reduced by treatment with ASA or indomethacin.
T10 990-1052 Sentence denotes This effect was reduced by treatment with ASA or indomethacin.
T11 1053-1198 Sentence denotes Prostaglandin production during the experiments was estimated by monitoring release of 6-keto-PGF1 alpha, the hydrolysis product of prostacyclin.
T11 1053-1198 Sentence denotes Prostaglandin production during the experiments was estimated by monitoring release of 6-keto-PGF1 alpha, the hydrolysis product of prostacyclin.
T12 1199-1364 Sentence denotes Abbreviation of time to arrhythmias by ligation was greatest in hearts that released large amounts of 6-keto-PGF1 alpha and least in those that released low amounts.
T12 1199-1364 Sentence denotes Abbreviation of time to arrhythmias by ligation was greatest in hearts that released large amounts of 6-keto-PGF1 alpha and least in those that released low amounts.
T13 1365-1513 Sentence denotes These results suggest that coronary ligation potentiates the arrhythmogenic effects of ouabain by a mechanism probably mediated by prostaglandin(s).
T13 1365-1513 Sentence denotes These results suggest that coronary ligation potentiates the arrhythmogenic effects of ouabain by a mechanism probably mediated by prostaglandin(s).