| Id |
Subject |
Object |
Predicate |
Lexical cue |
| T1 |
0-62 |
Sentence |
denotes |
Human dectin-1 deficiency and mucocutaneous fungal infections. |
| T1 |
0-62 |
Sentence |
denotes |
Human dectin-1 deficiency and mucocutaneous fungal infections. |
| T2 |
63-160 |
Sentence |
denotes |
Mucocutaneous fungal infections are typically found in patients who have no known immune defects. |
| T2 |
63-160 |
Sentence |
denotes |
Mucocutaneous fungal infections are typically found in patients who have no known immune defects. |
| T3 |
161-361 |
Sentence |
denotes |
We describe a family in which four women who were affected by either recurrent vulvovaginal candidiasis or onychomycosis had the early-stop-codon mutation Tyr238X in the beta-glucan receptor dectin-1. |
| T3 |
161-361 |
Sentence |
denotes |
We describe a family in which four women who were affected by either recurrent vulvovaginal candidiasis or onychomycosis had the early-stop-codon mutation Tyr238X in the beta-glucan receptor dectin-1. |
| T4 |
362-609 |
Sentence |
denotes |
The mutated form of dectin-1 was poorly expressed, did not mediate beta-glucan binding, and led to defective production of cytokines (interleukin-17, tumor necrosis factor, and interleukin-6) after stimulation with beta-glucan or Candida albicans. |
| T4 |
362-609 |
Sentence |
denotes |
The mutated form of dectin-1 was poorly expressed, did not mediate beta-glucan binding, and led to defective production of cytokines (interleukin-17, tumor necrosis factor, and interleukin-6) after stimulation with beta-glucan or Candida albicans. |
| T5 |
610-860 |
Sentence |
denotes |
In contrast, fungal phagocytosis and fungal killing were normal in the patients, explaining why dectin-1 deficiency was not associated with invasive fungal infections and highlighting the specific role of dectin-1 in human mucosal antifungal defense. |
| T5 |
610-860 |
Sentence |
denotes |
In contrast, fungal phagocytosis and fungal killing were normal in the patients, explaining why dectin-1 deficiency was not associated with invasive fungal infections and highlighting the specific role of dectin-1 in human mucosal antifungal defense. |
