| Id |
Subject |
Object |
Predicate |
Lexical cue |
| T1 |
0-85 |
Sentence |
denotes |
Cell activation and apoptosis by bacterial lipoproteins through toll-like receptor-2. |
| T1 |
0-85 |
Sentence |
denotes |
Cell activation and apoptosis by bacterial lipoproteins through toll-like receptor-2. |
| T2 |
86-194 |
Sentence |
denotes |
Apoptosis is implicated in the generation and resolution of inflammation in response to bacterial pathogens. |
| T2 |
86-194 |
Sentence |
denotes |
Apoptosis is implicated in the generation and resolution of inflammation in response to bacterial pathogens. |
| T3 |
195-289 |
Sentence |
denotes |
All bacterial pathogens produce lipoproteins (BLPs), which trigger the innate immune response. |
| T3 |
195-289 |
Sentence |
denotes |
All bacterial pathogens produce lipoproteins (BLPs), which trigger the innate immune response. |
| T4 |
290-394 |
Sentence |
denotes |
BLPs were found to induce apoptosis in THP-1 monocytic cells through human Toll-like receptor-2 (hTLR2). |
| T4 |
290-394 |
Sentence |
denotes |
BLPs were found to induce apoptosis in THP-1 monocytic cells through human Toll-like receptor-2 (hTLR2). |
| T5 |
395-475 |
Sentence |
denotes |
BLPs also initiated apoptosis in an epithelial cell line transfected with hTLR2. |
| T5 |
395-475 |
Sentence |
denotes |
BLPs also initiated apoptosis in an epithelial cell line transfected with hTLR2. |
| T6 |
476-638 |
Sentence |
denotes |
In addition, BLPs stimulated nuclear factor-kappaB, a transcriptional activator of multiple host defense genes, and activated the respiratory burst through hTLR2. |
| T6 |
476-638 |
Sentence |
denotes |
In addition, BLPs stimulated nuclear factor-kappaB, a transcriptional activator of multiple host defense genes, and activated the respiratory burst through hTLR2. |
| T7 |
639-738 |
Sentence |
denotes |
Thus, hTLR2 is a molecular link between microbial products, apoptosis, and host defense mechanisms. |
| T7 |
639-738 |
Sentence |
denotes |
Thus, hTLR2 is a molecular link between microbial products, apoptosis, and host defense mechanisms. |