PMC:17821 / 40653-53021 2 Projects
Annnotations
| Id | Subject | Object | Predicate | Lexical cue |
|---|---|---|---|---|
| T73 | 1170-1172 | 1605310 | denotes | 35 |
| T64 | 1556-1558 | 2456387 | denotes | 42 |
| T49 | 1996-1998 | 8214013 | denotes | 19 |
| T50 | 1999-2001 | 8814067 | denotes | 20 |
| T51 | 2002-2004 | 9797567 | denotes | 21 |
| T65 | 2038-2040 | 9632077 | denotes | 23 |
| T72 | 2277-2279 | 9797567 | denotes | 21 |
| T82 | 2280-2282 | 8317332 | denotes | 43 |
| T56 | 2598-2600 | 10366105 | denotes | 44 |
| T76 | 3872-3874 | 1605310 | denotes | 35 |
| T61 | 4124-4125 | 9461440 | denotes | 8 |
| T63 | 4677-4678 | 7621075 | denotes | 6 |
| T77 | 5040-5042 | 1605310 | denotes | 35 |
| T69 | 5043-5045 | 6501574 | denotes | 39 |
| T80 | 6675-6677 | 9808201 | denotes | 45 |
| T68 | 6861-6863 | 7401630 | denotes | 46 |
| T62 | 7875-7877 | 10623430 | denotes | 34 |
| T66 | 7875-7877 | 10623430 | denotes | 34 |
| T79 | 8118-8120 | 10366105 | denotes | 44 |
| T57 | 8711-8713 | 1828823 | denotes | 47 |
| T58 | 8714-8716 | 8157973 | denotes | 48 |
| T59 | 8790-8792 | 2784682 | denotes | 49 |
| T60 | 8793-8795 | 1578447 | denotes | 50 |
| T52 | 8843-8845 | 8569187 | denotes | 51 |
| T53 | 8846-8848 | 9742213 | denotes | 52 |
| T81 | 8922-8924 | 10366105 | denotes | 44 |
| T48 | 9047-9049 | 10037754 | denotes | 53 |
| T67 | 9048-9856 | 1605310 | denotes | 3]. Aggrecanase neoepitopes were abundantly found during the first phase of this arthritis [44]. Injection of zymosan into the knee joints of the three strains we studied was followed by a comparable depletion of PG; this finding suggests that the three strains have similar potencies to produce aggrecanase. Severe cartilage destruction, like chondrocyte death and cartilage erosion, was found only in DBA/1 mice. During the acute phase, PMNs constitute the dominant cell type present in the joint, whereas during the chronic phase, macrophages are the predominant cell type present in the synovium and synovial cavity. FcγR activation on both PMNs and macrophages may lead to the release of oxygen and nitrogen radicals, which in high concentrations can kill chondrocytes either by necrosis or apoptosis [5 |
| T78 | 9140-9142 | 10366105 | denotes | 44 |
| T70 | 9855-9857 | 7856740 | denotes | 54 |
| T71 | 9858-9860 | 10403282 | denotes | 55 |
| T74 | 10148-10150 | 1846914 | denotes | 56 |
| T75 | 10151-10153 | 7575726 | denotes | 57 |
| T55 | 10542-10874 | 9874574 | denotes | 1 joint. This attachment to the cartilage surface may cause release of enzymes that directly penetrate the cartilage matrix thus escaping from their inhibitors (manuscript in preparation). The present findings partly underline the findings in our previous study, in which an antigen-induced arthritis was produced in FcR γ-/-mice [5 |
| T54 | 10873-10875 | 10765918 | denotes | 58 |
