| Id |
Subject |
Object |
Predicate |
Lexical cue |
| BEL:20000014 |
878-1128 |
a(CHEBI:glucocorticoid) decreases p(MGI:Pomc) |
denotes |
Pulse-chase biosynthetic labeling studies showed that AtT-20 cells expressed much less RESP18 than the endogenous prohormone, POMC, but that glucocorticoid treatment lowered POMC and raised RESP18 biosynthetic rates so that they were nearly equimolar |
| BEL:20000016 |
878-1128 |
a(CHEBI:glucocorticoid) increases p(MGI:Resp18) |
denotes |
Pulse-chase biosynthetic labeling studies showed that AtT-20 cells expressed much less RESP18 than the endogenous prohormone, POMC, but that glucocorticoid treatment lowered POMC and raised RESP18 biosynthetic rates so that they were nearly equimolar |
| BEL:20000014 |
878-1128 |
a(CHEBI:glucocorticoid) decreases p(MGI:Pomc) |
denotes |
Pulse-chase biosynthetic labeling studies showed that AtT-20 cells expressed much less RESP18 than the endogenous prohormone, POMC, but that glucocorticoid treatment lowered POMC and raised RESP18 biosynthetic rates so that they were nearly equimolar |
| BEL:20000016 |
878-1128 |
a(CHEBI:glucocorticoid) increases p(MGI:Resp18) |
denotes |
Pulse-chase biosynthetic labeling studies showed that AtT-20 cells expressed much less RESP18 than the endogenous prohormone, POMC, but that glucocorticoid treatment lowered POMC and raised RESP18 biosynthetic rates so that they were nearly equimolar |
| BEL:20024020 |
878-1262 |
a(CHEBI:glucocorticoid) decreases p(MGI:Pomc) |
denotes |
Pulse-chase biosynthetic labeling studies showed that AtT-20 cells expressed much less RESP18 than the endogenous prohormone, POMC, but that glucocorticoid treatment lowered POMC and raised RESP18 biosynthetic rates so that they were nearly equimolar. Surprisingly, RESP18 was not processed to smaller peptides to any significant extent, nor was RESP18 or any smaller peptide secreted |
| BEL:20024022 |
878-1262 |
a(CHEBI:glucocorticoid) increases p(MGI:Resp18) |
denotes |
Pulse-chase biosynthetic labeling studies showed that AtT-20 cells expressed much less RESP18 than the endogenous prohormone, POMC, but that glucocorticoid treatment lowered POMC and raised RESP18 biosynthetic rates so that they were nearly equimolar. Surprisingly, RESP18 was not processed to smaller peptides to any significant extent, nor was RESP18 or any smaller peptide secreted |
