PubMed:23791840 / 0-559 JSONTXT

{"target":"https://pubannotation.org/docs/sourcedb/PubMed/sourceid/23791840","sourcedb":"PubMed","sourceid":"23791840","source_url":"https://www.ncbi.nlm.nih.gov/pubmed/23791840","text":"Superoxide dismutase 1 overexpression in mice abolishes maternal diabetes-induced endoplasmic reticulum stress in diabetic embryopathy.\nOBJECTIVE: Both oxidative stress and endoplasmic reticulum stress (ER stress) are causal events in diabetic embryopathy. We tested whether oxidative stress causes ER stress.\nSTUDY DESIGN: Wild-type (WT) and superoxide dismutase 1 (SOD1)-overexpressing day 8.75 embryos from nondiabetic WT control with SOD1 transgenic male and diabetic WT female with SOD1 transgenic male were analyzed for ER stress markers: 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