PubMed:23609450 / 1366-1580 JSONTXT

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    sentences

    {"project":"sentences","denotations":[{"id":"T9","span":{"begin":0,"end":214},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Our results demonstrate that inducible IRG1 promotes endotoxin tolerance by increasing A20 expression through ROS, indicating a new molecular mechanism regulating hypoinflammation of sepsis and endotoxin tolerance."}

    Inflammaging

    {"project":"Inflammaging","denotations":[{"id":"T9","span":{"begin":0,"end":214},"obj":"Sentence"},{"id":"T9","span":{"begin":0,"end":214},"obj":"Sentence"}],"text":"Our results demonstrate that inducible IRG1 promotes endotoxin tolerance by increasing A20 expression through ROS, indicating a new molecular mechanism regulating hypoinflammation of sepsis and endotoxin tolerance."}

    PubmedHPO

    {"project":"PubmedHPO","denotations":[{"id":"T2","span":{"begin":183,"end":189},"obj":"HP_0100806"}],"text":"Our results demonstrate that inducible IRG1 promotes endotoxin tolerance by increasing A20 expression through ROS, indicating a new molecular mechanism regulating hypoinflammation of sepsis and endotoxin tolerance."}

    DisGeNET5_gene_disease

    {"project":"DisGeNET5_gene_disease","denotations":[{"id":"23609450-8#39#43#gene730249","span":{"begin":39,"end":43},"obj":"gene730249"},{"id":"23609450-8#183#189#diseaseC0036690","span":{"begin":183,"end":189},"obj":"diseaseC0036690"},{"id":"23609450-8#183#189#diseaseC0243026","span":{"begin":183,"end":189},"obj":"diseaseC0243026"}],"relations":[{"id":"39#43#gene730249183#189#diseaseC0036690","pred":"associated_with","subj":"23609450-8#39#43#gene730249","obj":"23609450-8#183#189#diseaseC0036690"},{"id":"39#43#gene730249183#189#diseaseC0243026","pred":"associated_with","subj":"23609450-8#39#43#gene730249","obj":"23609450-8#183#189#diseaseC0243026"}],"text":"Our results demonstrate that inducible IRG1 promotes endotoxin tolerance by increasing A20 expression through ROS, indicating a new molecular mechanism regulating hypoinflammation of sepsis and endotoxin tolerance."}

    PubCasesHPO

    {"project":"PubCasesHPO","denotations":[{"id":"AB2","span":{"begin":183,"end":189},"obj":"HP:0100806"}],"text":"Our results demonstrate that inducible IRG1 promotes endotoxin tolerance by increasing A20 expression through ROS, indicating a new molecular mechanism regulating hypoinflammation of sepsis and endotoxin tolerance."}

    HP-phenotype

    {"project":"HP-phenotype","denotations":[{"id":"T3","span":{"begin":183,"end":189},"obj":"Phenotype"}],"attributes":[{"id":"A3","pred":"hp_id","subj":"T3","obj":"HP:0100806"}],"namespaces":[{"prefix":"HP","uri":"http://purl.obolibrary.org/obo/HP_"}],"text":"Our results demonstrate that inducible IRG1 promotes endotoxin tolerance by increasing A20 expression through ROS, indicating a new molecular mechanism regulating hypoinflammation of sepsis and endotoxin tolerance."}

    NCBITAXON

    {"project":"NCBITAXON","denotations":[{"id":"T4","span":{"begin":183,"end":189},"obj":"OrganismTaxon"}],"attributes":[{"id":"A4","pred":"db_id","subj":"T4","obj":"137507"}],"text":"Our results demonstrate that inducible IRG1 promotes endotoxin tolerance by increasing A20 expression through ROS, indicating a new molecular mechanism regulating hypoinflammation of sepsis and endotoxin tolerance."}