PubMed:20949073 / 0-153 JSONTXT

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    LitCoin-sentences

    {"project":"LitCoin-sentences","denotations":[{"id":"T1","span":{"begin":0,"end":77},"obj":"Sentence"}],"text":"Alternative splicing at a NAGNAG acceptor site as a novel phenotype modifier.\nApproximately 30% of alleles causing genetic disorders generate premature t"}

    LitCoin-GeneOrGeneProduct-v0

    {"project":"LitCoin-GeneOrGeneProduct-v0","denotations":[{"id":"T1","span":{"begin":12,"end":20},"obj":"GeneOrGeneProduct"},{"id":"T2","span":{"begin":52,"end":57},"obj":"GeneOrGeneProduct"},{"id":"T3","span":{"begin":78,"end":91},"obj":"GeneOrGeneProduct"}],"text":"Alternative splicing at a NAGNAG acceptor site as a novel phenotype modifier.\nApproximately 30% of alleles causing genetic disorders generate premature t"}

    LitCoin-GeneOrGeneProduct-v2

    {"project":"LitCoin-GeneOrGeneProduct-v2","denotations":[{"id":"T1","span":{"begin":52,"end":57},"obj":"GeneOrGeneProduct"}],"text":"Alternative splicing at a NAGNAG acceptor site as a novel phenotype modifier.\nApproximately 30% of alleles causing genetic disorders generate premature t"}

    LitCoin-Disease-MeSH

    {"project":"LitCoin-Disease-MeSH","denotations":[{"id":"T1","span":{"begin":115,"end":132},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A1","pred":"originalLabel","subj":"T1","obj":"D030342"}],"text":"Alternative splicing at a NAGNAG acceptor site as a novel phenotype modifier.\nApproximately 30% of alleles causing genetic disorders generate premature t"}

    LitCoin_Mondo_095

    {"project":"LitCoin_Mondo_095","denotations":[{"id":"T1","span":{"begin":115,"end":132},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A1","pred":"mondo_id","subj":"T1","obj":"0003847"}],"text":"Alternative splicing at a NAGNAG acceptor site as a novel phenotype modifier.\nApproximately 30% of alleles causing genetic disorders generate premature t"}

    LitCoin-MeSH-Disease-2

    {"project":"LitCoin-MeSH-Disease-2","denotations":[{"id":"T1","span":{"begin":115,"end":132},"obj":"DiseaseOrPhenotypicFeature"}],"attributes":[{"id":"A1","pred":"ID:","subj":"T1","obj":"D030342"}],"text":"Alternative splicing at a NAGNAG acceptor site as a novel phenotype modifier.\nApproximately 30% of alleles causing genetic disorders generate premature t"}