PubMed:20172938 / 1162-1684 JSONTXT

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{"target":"https://pubannotation.org/docs/sourcedb/PubMed/sourceid/20172938","sourcedb":"PubMed","sourceid":"20172938","text":"Ischaemia led to a significant (P\u003c0.05) increase by 70% in transudate formation during reperfusion only in hearts without sevoflurane. This was accompanied by significant (P\u003c0.05) increases in heparan sulphate (four-fold) and syndecan release (6.5-fold), with electron microscopy revealing massive degradation of glycocalyx. After ischaemia, histamine was released into transudate, and cathepsin B activity increased in effluent (P\u003c0.05). 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