PubMed:14597618 / 1120-1341 JSONTXT

{"project":"sentences","denotations":[{"id":"T9","span":{"begin":0,"end":221},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Cardiac hypertrophy induced by overexpression of IGF1R was completely blocked by a dominant negative PI3K(p110alpha) mutant, suggesting IGF1R promotes compensated cardiac hypertrophy in a PI3K(p110alpha)-dependent manner."}

{"project":"2015-BEL-Sample-2","denotations":[{"id":"BEL:20038198","span":{"begin":0,"end":220},"obj":"kin(p(MGI:Igf1r)) increases kin(p(MGI:Pik3ca))"}],"text":"Cardiac hypertrophy induced by overexpression of IGF1R was completely blocked by a dominant negative PI3K(p110alpha) mutant, suggesting IGF1R promotes compensated cardiac hypertrophy in a PI3K(p110alpha)-dependent manner."}