Id |
Subject |
Object |
Predicate |
Lexical cue |
T1 |
0-79 |
Sentence |
denotes |
Hepatitis type C virus infection in patients with type B chronic liver disease. |
T2 |
80-269 |
Sentence |
denotes |
Anti-c100-3 (Ortho) was determined in the sera of 152 patients with HBs antigen-positive chronic liver diseases to assess coinfection of hepatitis B virus (HBV) and hepatitis C virus (HCV). |
T3 |
270-323 |
Sentence |
denotes |
Eleven patients (7.2%) were positive for anti-c100-3. |
T4 |
324-405 |
Sentence |
denotes |
Anti-CP-9 (Okamoto) and HCV-RNA (RT-PCR) were also examined in these 11 patients. |
T5 |
406-487 |
Sentence |
denotes |
Anti-CP-9 was detected in 7 patients and HCV-RNA was detected in all 11 patients. |
T6 |
488-596 |
Sentence |
denotes |
Four of the 11 anti-c100-3-positive patients were positive for HBe antigen (HBeAg) and others were negative. |
T7 |
597-681 |
Sentence |
denotes |
In 8 of the 11 patients, HCV was suspected to be superinfected by blood transfusion. |
T8 |
682-840 |
Sentence |
denotes |
In HBeAg-positive patients, serum glutamic pyruvic transaminase (SGPT) was elevated in relation to active replication of HBV shown by DNA-polymerase activity. |
T9 |
841-926 |
Sentence |
denotes |
The histological findings showed chronic active hepatitis, with or without cirrhosis. |
T10 |
927-1036 |
Sentence |
denotes |
On the other hand, in HBeAg-negative patients, SGPT fluctuated without evidence of active replication of HBV. |
T11 |
1037-1133 |
Sentence |
denotes |
Active inflammation in the liver was observed in 3 of 5 HBeAg-negative patients by liver biopsy. |
T12 |
1134-1383 |
Sentence |
denotes |
These findings suggest that HBV might play an important role in chronic active inflammation in HBeAg-positive patients coinfected with HCV, and that HCV might be responsible for continuous inflammation in HBeAg-negative patients coinfected with HCV. |
T1 |
0-79 |
Sentence |
denotes |
Hepatitis type C virus infection in patients with type B chronic liver disease. |
T2 |
80-269 |
Sentence |
denotes |
Anti-c100-3 (Ortho) was determined in the sera of 152 patients with HBs antigen-positive chronic liver diseases to assess coinfection of hepatitis B virus (HBV) and hepatitis C virus (HCV). |
T3 |
270-323 |
Sentence |
denotes |
Eleven patients (7.2%) were positive for anti-c100-3. |
T4 |
324-405 |
Sentence |
denotes |
Anti-CP-9 (Okamoto) and HCV-RNA (RT-PCR) were also examined in these 11 patients. |
T5 |
406-487 |
Sentence |
denotes |
Anti-CP-9 was detected in 7 patients and HCV-RNA was detected in all 11 patients. |
T6 |
488-596 |
Sentence |
denotes |
Four of the 11 anti-c100-3-positive patients were positive for HBe antigen (HBeAg) and others were negative. |
T7 |
597-681 |
Sentence |
denotes |
In 8 of the 11 patients, HCV was suspected to be superinfected by blood transfusion. |
T8 |
682-840 |
Sentence |
denotes |
In HBeAg-positive patients, serum glutamic pyruvic transaminase (SGPT) was elevated in relation to active replication of HBV shown by DNA-polymerase activity. |
T9 |
841-926 |
Sentence |
denotes |
The histological findings showed chronic active hepatitis, with or without cirrhosis. |
T10 |
927-1036 |
Sentence |
denotes |
On the other hand, in HBeAg-negative patients, SGPT fluctuated without evidence of active replication of HBV. |
T11 |
1037-1133 |
Sentence |
denotes |
Active inflammation in the liver was observed in 3 of 5 HBeAg-negative patients by liver biopsy. |
T12 |
1134-1383 |
Sentence |
denotes |
These findings suggest that HBV might play an important role in chronic active inflammation in HBeAg-positive patients coinfected with HCV, and that HCV might be responsible for continuous inflammation in HBeAg-negative patients coinfected with HCV. |