PMC:7565665 / 39585-39853 JSONTXT

{"project":"LitCovid-PD-FMA-UBERON","denotations":[{"id":"T261","span":{"begin":67,"end":71},"obj":"Body_part"},{"id":"T262","span":{"begin":187,"end":191},"obj":"Body_part"},{"id":"T263","span":{"begin":241,"end":245},"obj":"Body_part"}],"attributes":[{"id":"A261","pred":"fma_id","subj":"T261","obj":"http://purl.org/sig/ont/fma/fma7195"},{"id":"A262","pred":"fma_id","subj":"T262","obj":"http://purl.org/sig/ont/fma/fma7195"},{"id":"A263","pred":"fma_id","subj":"T263","obj":"http://purl.org/sig/ont/fma/fma7195"}],"text":"Similarly, in mice deficient in LC3B (LC3B−/−), bleomycin-mediated lung injury and fibrotic changes were more pronounced, suggesting the protective role of autophagy in bleomycin-induced lung injury and the resulting development of fibrotic lung disease in mice [157]."}

{"project":"LitCovid-PD-UBERON","denotations":[{"id":"T70","span":{"begin":67,"end":71},"obj":"Body_part"},{"id":"T71","span":{"begin":187,"end":191},"obj":"Body_part"},{"id":"T72","span":{"begin":241,"end":245},"obj":"Body_part"}],"attributes":[{"id":"A70","pred":"uberon_id","subj":"T70","obj":"http://purl.obolibrary.org/obo/UBERON_0002048"},{"id":"A71","pred":"uberon_id","subj":"T71","obj":"http://purl.obolibrary.org/obo/UBERON_0002048"},{"id":"A72","pred":"uberon_id","subj":"T72","obj":"http://purl.obolibrary.org/obo/UBERON_0002048"}],"text":"Similarly, in mice deficient in LC3B (LC3B−/−), bleomycin-mediated lung injury and fibrotic changes were more pronounced, suggesting the protective role of autophagy in bleomycin-induced lung injury and the resulting development of fibrotic lung disease in mice [157]."}

{"project":"LitCovid-PD-MONDO","denotations":[{"id":"T324","span":{"begin":72,"end":78},"obj":"Disease"},{"id":"T325","span":{"begin":192,"end":198},"obj":"Disease"},{"id":"T326","span":{"begin":241,"end":253},"obj":"Disease"}],"attributes":[{"id":"A324","pred":"mondo_id","subj":"T324","obj":"http://purl.obolibrary.org/obo/MONDO_0021178"},{"id":"A325","pred":"mondo_id","subj":"T325","obj":"http://purl.obolibrary.org/obo/MONDO_0021178"},{"id":"A326","pred":"mondo_id","subj":"T326","obj":"http://purl.obolibrary.org/obo/MONDO_0005275"}],"text":"Similarly, in mice deficient in LC3B (LC3B−/−), bleomycin-mediated lung injury and fibrotic changes were more pronounced, suggesting the protective role of autophagy in bleomycin-induced lung injury and the resulting development of fibrotic lung disease in mice [157]."}

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{"project":"LitCovid-PD-CHEBI","denotations":[{"id":"T201","span":{"begin":48,"end":57},"obj":"Chemical"},{"id":"T202","span":{"begin":169,"end":178},"obj":"Chemical"}],"attributes":[{"id":"A201","pred":"chebi_id","subj":"T201","obj":"http://purl.obolibrary.org/obo/CHEBI_22907"},{"id":"A202","pred":"chebi_id","subj":"T202","obj":"http://purl.obolibrary.org/obo/CHEBI_22907"}],"text":"Similarly, in mice deficient in LC3B (LC3B−/−), bleomycin-mediated lung injury and fibrotic changes were more pronounced, suggesting the protective role of autophagy in bleomycin-induced lung injury and the resulting development of fibrotic lung disease in mice [157]."}

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{"project":"LitCovid-PD-HP","denotations":[{"id":"T139","span":{"begin":241,"end":253},"obj":"Phenotype"}],"attributes":[{"id":"A139","pred":"hp_id","subj":"T139","obj":"http://purl.obolibrary.org/obo/HP_0002088"}],"text":"Similarly, in mice deficient in LC3B (LC3B−/−), bleomycin-mediated lung injury and fibrotic changes were more pronounced, suggesting the protective role of autophagy in bleomycin-induced lung injury and the resulting development of fibrotic lung disease in mice [157]."}

{"project":"LitCovid-PD-GO-BP","denotations":[{"id":"T523","span":{"begin":156,"end":165},"obj":"http://purl.obolibrary.org/obo/GO_0016236"},{"id":"T524","span":{"begin":156,"end":165},"obj":"http://purl.obolibrary.org/obo/GO_0006914"}],"text":"Similarly, in mice deficient in LC3B (LC3B−/−), bleomycin-mediated lung injury and fibrotic changes were more pronounced, suggesting the protective role of autophagy in bleomycin-induced lung injury and the resulting development of fibrotic lung disease in mice [157]."}

{"project":"LitCovid-sentences","denotations":[{"id":"T209","span":{"begin":0,"end":268},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"Similarly, in mice deficient in LC3B (LC3B−/−), bleomycin-mediated lung injury and fibrotic changes were more pronounced, suggesting the protective role of autophagy in bleomycin-induced lung injury and the resulting development of fibrotic lung disease in mice [157]."}