PMC:7546716 / 6234-6592 JSONTXT

{"project":"LitCovid-PD-FMA-UBERON","denotations":[{"id":"T91","span":{"begin":14,"end":16},"obj":"Body_part"},{"id":"T92","span":{"begin":73,"end":75},"obj":"Body_part"},{"id":"T93","span":{"begin":87,"end":89},"obj":"Body_part"},{"id":"T94","span":{"begin":133,"end":137},"obj":"Body_part"},{"id":"T95","span":{"begin":138,"end":142},"obj":"Body_part"},{"id":"T96","span":{"begin":150,"end":155},"obj":"Body_part"},{"id":"T97","span":{"begin":252,"end":257},"obj":"Body_part"},{"id":"T98","span":{"begin":284,"end":288},"obj":"Body_part"}],"attributes":[{"id":"A91","pred":"fma_id","subj":"T91","obj":"http://purl.org/sig/ont/fma/fma86578"},{"id":"A92","pred":"fma_id","subj":"T92","obj":"http://purl.org/sig/ont/fma/fma86578"},{"id":"A93","pred":"fma_id","subj":"T93","obj":"http://purl.org/sig/ont/fma/fma86578"},{"id":"A94","pred":"fma_id","subj":"T94","obj":"http://purl.org/sig/ont/fma/fma68646"},{"id":"A95","pred":"fma_id","subj":"T95","obj":"http://purl.org/sig/ont/fma/fma12520"},{"id":"A96","pred":"fma_id","subj":"T96","obj":"http://purl.org/sig/ont/fma/fma68877"},{"id":"A97","pred":"fma_id","subj":"T97","obj":"http://purl.org/sig/ont/fma/fma68646"},{"id":"A98","pred":"fma_id","subj":"T98","obj":"http://purl.org/sig/ont/fma/fma7195"}],"text":"ant source of IL-9.20 By acting in both autocrine and paracrine manners, IL-33-induced IL-9 might sustain a proinflammatory ILC2–γδT cell axis in the lungs of patients with COVID-19, thus initiating mild–moderate forms of pneumonia.\nBoth ILC2 and γδ T cells are centrally involved in lung homoeostasis and are rapidly activated in response to pathogens inclu"}

{"project":"LitCovid-PD-UBERON","denotations":[{"id":"T9","span":{"begin":284,"end":288},"obj":"Body_part"}],"attributes":[{"id":"A9","pred":"uberon_id","subj":"T9","obj":"http://purl.obolibrary.org/obo/UBERON_0002048"}],"text":"ant source of IL-9.20 By acting in both autocrine and paracrine manners, IL-33-induced IL-9 might sustain a proinflammatory ILC2–γδT cell axis in the lungs of patients with COVID-19, thus initiating mild–moderate forms of pneumonia.\nBoth ILC2 and γδ T cells are centrally involved in lung homoeostasis and are rapidly activated in response to pathogens inclu"}

{"project":"LitCovid-PD-MONDO","denotations":[{"id":"T55","span":{"begin":173,"end":181},"obj":"Disease"},{"id":"T56","span":{"begin":222,"end":231},"obj":"Disease"}],"attributes":[{"id":"A55","pred":"mondo_id","subj":"T55","obj":"http://purl.obolibrary.org/obo/MONDO_0100096"},{"id":"A56","pred":"mondo_id","subj":"T56","obj":"http://purl.obolibrary.org/obo/MONDO_0005249"}],"text":"ant source of IL-9.20 By acting in both autocrine and paracrine manners, IL-33-induced IL-9 might sustain a proinflammatory ILC2–γδT cell axis in the lungs of patients with COVID-19, thus initiating mild–moderate forms of pneumonia.\nBoth ILC2 and γδ T cells are centrally involved in lung homoeostasis and are rapidly activated in response to pathogens inclu"}

{"project":"LitCovid-PD-CLO","denotations":[{"id":"T78","span":{"begin":106,"end":107},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"},{"id":"T79","span":{"begin":131,"end":137},"obj":"http://purl.obolibrary.org/obo/CL_0000084"},{"id":"T80","span":{"begin":150,"end":155},"obj":"http://www.ebi.ac.uk/efo/EFO_0000934"},{"id":"T81","span":{"begin":250,"end":257},"obj":"http://purl.obolibrary.org/obo/CL_0000084"},{"id":"T82","span":{"begin":284,"end":288},"obj":"http://purl.obolibrary.org/obo/UBERON_0002048"},{"id":"T83","span":{"begin":284,"end":288},"obj":"http://www.ebi.ac.uk/efo/EFO_0000934"},{"id":"T84","span":{"begin":318,"end":327},"obj":"http://purl.obolibrary.org/obo/CLO_0001658"}],"text":"ant source of IL-9.20 By acting in both autocrine and paracrine manners, IL-33-induced IL-9 might sustain a proinflammatory ILC2–γδT cell axis in the lungs of patients with COVID-19, thus initiating mild–moderate forms of pneumonia.\nBoth ILC2 and γδ T cells are centrally involved in lung homoeostasis and are rapidly activated in response to pathogens inclu"}

{"project":"LitCovid-PD-CHEBI","denotations":[{"id":"T65","span":{"begin":14,"end":16},"obj":"Chemical"},{"id":"T67","span":{"begin":73,"end":75},"obj":"Chemical"},{"id":"T69","span":{"begin":87,"end":89},"obj":"Chemical"}],"attributes":[{"id":"A65","pred":"chebi_id","subj":"T65","obj":"http://purl.obolibrary.org/obo/CHEBI_63895"},{"id":"A66","pred":"chebi_id","subj":"T65","obj":"http://purl.obolibrary.org/obo/CHEBI_74072"},{"id":"A67","pred":"chebi_id","subj":"T67","obj":"http://purl.obolibrary.org/obo/CHEBI_63895"},{"id":"A68","pred":"chebi_id","subj":"T67","obj":"http://purl.obolibrary.org/obo/CHEBI_74072"},{"id":"A69","pred":"chebi_id","subj":"T69","obj":"http://purl.obolibrary.org/obo/CHEBI_63895"},{"id":"A70","pred":"chebi_id","subj":"T69","obj":"http://purl.obolibrary.org/obo/CHEBI_74072"}],"text":"ant source of IL-9.20 By acting in both autocrine and paracrine manners, IL-33-induced IL-9 might sustain a proinflammatory ILC2–γδT cell axis in the lungs of patients with COVID-19, thus initiating mild–moderate forms of pneumonia.\nBoth ILC2 and γδ T cells are centrally involved in lung homoeostasis and are rapidly activated in response to pathogens inclu"}

{"project":"LitCovid-PD-HP","denotations":[{"id":"T10","span":{"begin":222,"end":231},"obj":"Phenotype"}],"attributes":[{"id":"A10","pred":"hp_id","subj":"T10","obj":"http://purl.obolibrary.org/obo/HP_0002090"}],"text":"ant source of IL-9.20 By acting in both autocrine and paracrine manners, IL-33-induced IL-9 might sustain a proinflammatory ILC2–γδT cell axis in the lungs of patients with COVID-19, thus initiating mild–moderate forms of pneumonia.\nBoth ILC2 and γδ T cells are centrally involved in lung homoeostasis and are rapidly activated in response to pathogens inclu"}

{"project":"LitCovid-PubTator","denotations":[{"id":"226","span":{"begin":14,"end":18},"obj":"Gene"},{"id":"227","span":{"begin":73,"end":78},"obj":"Gene"},{"id":"228","span":{"begin":87,"end":91},"obj":"Gene"},{"id":"231","span":{"begin":159,"end":167},"obj":"Species"},{"id":"238","span":{"begin":173,"end":181},"obj":"Disease"},{"id":"239","span":{"begin":222,"end":231},"obj":"Disease"},{"id":"370","span":{"begin":284,"end":301},"obj":"Disease"}],"attributes":[{"id":"A226","pred":"tao:has_database_id","subj":"226","obj":"Gene:3578"},{"id":"A227","pred":"tao:has_database_id","subj":"227","obj":"Gene:90865"},{"id":"A228","pred":"tao:has_database_id","subj":"228","obj":"Gene:3578"},{"id":"A231","pred":"tao:has_database_id","subj":"231","obj":"Tax:9606"},{"id":"A238","pred":"tao:has_database_id","subj":"238","obj":"MESH:C000657245"},{"id":"A239","pred":"tao:has_database_id","subj":"239","obj":"MESH:D011014"},{"id":"A370","pred":"tao:has_database_id","subj":"370","obj":"MESH:D008171"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"ant source of IL-9.20 By acting in both autocrine and paracrine manners, IL-33-induced IL-9 might sustain a proinflammatory ILC2–γδT cell axis in the lungs of patients with COVID-19, thus initiating mild–moderate forms of pneumonia.\nBoth ILC2 and γδ T cells are centrally involved in lung homoeostasis and are rapidly activated in response to pathogens inclu"}

{"project":"2_test","denotations":[{"id":"33073244-27791087-66244525","span":{"begin":19,"end":21},"obj":"27791087"}],"text":"ant source of IL-9.20 By acting in both autocrine and paracrine manners, IL-33-induced IL-9 might sustain a proinflammatory ILC2–γδT cell axis in the lungs of patients with COVID-19, thus initiating mild–moderate forms of pneumonia.\nBoth ILC2 and γδ T cells are centrally involved in lung homoeostasis and are rapidly activated in response to pathogens inclu"}