ant source of IL-9.20 By acting in both autocrine and paracrine manners, IL-33-induced IL-9 might sustain a proinflammatory ILC2–γδT cell axis in the lungs of patients with COVID-19, thus initiating mild–moderate forms of pneumonia.
Both ILC2 and γδ T cells are centrally involved in lung homoeostasis and are rapidly activated in response to pathogens inclu