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PMC:7519301 / 32112-33015 JSONTXT

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LitCovid-PD-MONDO

Id Subject Object Predicate Lexical cue mondo_id
T161 192-200 Disease denotes SARS-CoV http://purl.obolibrary.org/obo/MONDO_0005091
T162 192-196 Disease denotes SARS http://purl.obolibrary.org/obo/MONDO_0005091
T163 513-523 Disease denotes infections http://purl.obolibrary.org/obo/MONDO_0005550

LitCovid-PD-CLO

Id Subject Object Predicate Lexical cue
T258 124-125 http://purl.obolibrary.org/obo/CLO_0001020 denotes A
T259 321-322 http://purl.obolibrary.org/obo/CLO_0001020 denotes a
T260 323-328 http://purl.obolibrary.org/obo/NCBITaxon_10239 denotes virus
T261 490-491 http://purl.obolibrary.org/obo/CLO_0001020 denotes a

LitCovid-PubTator

Id Subject Object Predicate Lexical cue tao:has_database_id tao:has_standard_notation
463 192-202 Species denotes SARS-CoV-2 Tax:2697049
464 513-523 Disease denotes infections MESH:D007239
465 888-893 Mutation denotes D614G p.D614G
466 342-347 Mutation denotes D614G p.D614G
467 117-122 Mutation denotes D614G p.D614G

LitCovid-sentences

Id Subject Object Predicate Lexical cue
T194 0-123 Sentence denotes These data indicate that epidemiologic factors could be sufficient to explain the global spread of mutations such as D614G.
T195 124-290 Sentence denotes A founder effect means that these mutations were likely exported to SARS-CoV-2 naive areas early in the outbreak and therefore given the opportunity to spread widely.
T196 291-427 Sentence denotes As such, on January 28, 2020, a virus carrying the D614G mutation, which was rare among sequences from China, was identified in Germany.
T197 428-631 Sentence denotes Host and environmental factors permitted the establishment of a sustained cluster of infections that propagated this mutation until it became dominant among European sequences and then globally (Fig. 2).
T198 632-794 Sentence denotes We found no evidence that the frequent identification of this mutation was caused by convergent selection events that would have occurred in multiple individuals.
T199 795-903 Sentence denotes Further analyses are needed to characterize the biologic mechanisms behind the spread of the D614G mutation.