PMC:7373848 / 31128-31879 JSONTXT

Annnotations TAB JSON ListView MergeView

    LitCovid-PD-FMA-UBERON

    {"project":"LitCovid-PD-FMA-UBERON","denotations":[{"id":"T500","span":{"begin":170,"end":182},"obj":"Body_part"},{"id":"T501","span":{"begin":217,"end":222},"obj":"Body_part"},{"id":"T502","span":{"begin":226,"end":234},"obj":"Body_part"},{"id":"T503","span":{"begin":405,"end":421},"obj":"Body_part"},{"id":"T504","span":{"begin":417,"end":421},"obj":"Body_part"},{"id":"T505","span":{"begin":582,"end":596},"obj":"Body_part"},{"id":"T506","span":{"begin":645,"end":655},"obj":"Body_part"}],"attributes":[{"id":"A500","pred":"fma_id","subj":"T500","obj":"http://purl.org/sig/ont/fma/fma63183"},{"id":"A501","pred":"fma_id","subj":"T501","obj":"http://purl.org/sig/ont/fma/fma63814"},{"id":"A502","pred":"fma_id","subj":"T502","obj":"http://purl.org/sig/ont/fma/fma63812"},{"id":"A503","pred":"fma_id","subj":"T503","obj":"http://purl.org/sig/ont/fma/fma66772"},{"id":"A504","pred":"fma_id","subj":"T504","obj":"http://purl.org/sig/ont/fma/fma68646"},{"id":"A505","pred":"fma_id","subj":"T505","obj":"http://purl.org/sig/ont/fma/fma62871"},{"id":"A506","pred":"fma_id","subj":"T506","obj":"http://purl.org/sig/ont/fma/fma62863"}],"text":"A discussion of vascular disease should distinguish causal, pathological, structural and functional elements. The term “vasculopathy” represents any abnormality within a blood vessel- small, medium and large caliber, veins or arteries (reviewed in Seshan) [85]. Degenerative, metabolic and inflammatory conditions, embolic diseases and coagulation disorders are responsible for acute, subacute or chronic endothelial cell injury. In many cases, non-inflammatory (necrotizing) vascular injury causes accumulation of immune complexes within the vessel’s wall. A thickened intima with immunoglobulin and complement deposition, without inflammatory lymphocyte infiltration or atherosclerotic change are typical features of an autoimmune vasculopathy [86]."}

    LitCovid-PD-UBERON

    {"project":"LitCovid-PD-UBERON","denotations":[{"id":"T138","span":{"begin":170,"end":182},"obj":"Body_part"},{"id":"T139","span":{"begin":170,"end":175},"obj":"Body_part"},{"id":"T140","span":{"begin":176,"end":182},"obj":"Body_part"}],"attributes":[{"id":"A138","pred":"uberon_id","subj":"T138","obj":"http://purl.obolibrary.org/obo/UBERON_0001981"},{"id":"A139","pred":"uberon_id","subj":"T139","obj":"http://purl.obolibrary.org/obo/UBERON_0000178"},{"id":"A140","pred":"uberon_id","subj":"T140","obj":"http://purl.obolibrary.org/obo/UBERON_0000055"}],"text":"A discussion of vascular disease should distinguish causal, pathological, structural and functional elements. The term “vasculopathy” represents any abnormality within a blood vessel- small, medium and large caliber, veins or arteries (reviewed in Seshan) [85]. Degenerative, metabolic and inflammatory conditions, embolic diseases and coagulation disorders are responsible for acute, subacute or chronic endothelial cell injury. In many cases, non-inflammatory (necrotizing) vascular injury causes accumulation of immune complexes within the vessel’s wall. A thickened intima with immunoglobulin and complement deposition, without inflammatory lymphocyte infiltration or atherosclerotic change are typical features of an autoimmune vasculopathy [86]."}

    LitCovid-PubTator

    {"project":"LitCovid-PubTator","denotations":[{"id":"631","span":{"begin":16,"end":32},"obj":"Disease"},{"id":"632","span":{"begin":120,"end":132},"obj":"Disease"},{"id":"633","span":{"begin":315,"end":331},"obj":"Disease"},{"id":"634","span":{"begin":336,"end":357},"obj":"Disease"},{"id":"635","span":{"begin":476,"end":491},"obj":"Disease"},{"id":"636","span":{"begin":672,"end":694},"obj":"Disease"},{"id":"637","span":{"begin":722,"end":745},"obj":"Disease"}],"attributes":[{"id":"A631","pred":"tao:has_database_id","subj":"631","obj":"MESH:D000783"},{"id":"A632","pred":"tao:has_database_id","subj":"632","obj":"MESH:D020144"},{"id":"A633","pred":"tao:has_database_id","subj":"633","obj":"MESH:D004617"},{"id":"A634","pred":"tao:has_database_id","subj":"634","obj":"MESH:D001778"},{"id":"A635","pred":"tao:has_database_id","subj":"635","obj":"MESH:D057772"},{"id":"A636","pred":"tao:has_database_id","subj":"636","obj":"MESH:D050197"},{"id":"A637","pred":"tao:has_database_id","subj":"637","obj":"MESH:D001327"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"A discussion of vascular disease should distinguish causal, pathological, structural and functional elements. The term “vasculopathy” represents any abnormality within a blood vessel- small, medium and large caliber, veins or arteries (reviewed in Seshan) [85]. Degenerative, metabolic and inflammatory conditions, embolic diseases and coagulation disorders are responsible for acute, subacute or chronic endothelial cell injury. In many cases, non-inflammatory (necrotizing) vascular injury causes accumulation of immune complexes within the vessel’s wall. A thickened intima with immunoglobulin and complement deposition, without inflammatory lymphocyte infiltration or atherosclerotic change are typical features of an autoimmune vasculopathy [86]."}

    LitCovid-PD-MONDO

    {"project":"LitCovid-PD-MONDO","denotations":[{"id":"T166","span":{"begin":16,"end":32},"obj":"Disease"},{"id":"T167","span":{"begin":336,"end":357},"obj":"Disease"},{"id":"T168","span":{"begin":422,"end":428},"obj":"Disease"},{"id":"T169","span":{"begin":485,"end":491},"obj":"Disease"}],"attributes":[{"id":"A166","pred":"mondo_id","subj":"T166","obj":"http://purl.obolibrary.org/obo/MONDO_0005385"},{"id":"A167","pred":"mondo_id","subj":"T167","obj":"http://purl.obolibrary.org/obo/MONDO_0001531"},{"id":"A168","pred":"mondo_id","subj":"T168","obj":"http://purl.obolibrary.org/obo/MONDO_0021178"},{"id":"A169","pred":"mondo_id","subj":"T169","obj":"http://purl.obolibrary.org/obo/MONDO_0021178"}],"text":"A discussion of vascular disease should distinguish causal, pathological, structural and functional elements. The term “vasculopathy” represents any abnormality within a blood vessel- small, medium and large caliber, veins or arteries (reviewed in Seshan) [85]. Degenerative, metabolic and inflammatory conditions, embolic diseases and coagulation disorders are responsible for acute, subacute or chronic endothelial cell injury. In many cases, non-inflammatory (necrotizing) vascular injury causes accumulation of immune complexes within the vessel’s wall. A thickened intima with immunoglobulin and complement deposition, without inflammatory lymphocyte infiltration or atherosclerotic change are typical features of an autoimmune vasculopathy [86]."}

    LitCovid-PD-CLO

    {"project":"LitCovid-PD-CLO","denotations":[{"id":"T510","span":{"begin":0,"end":1},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"},{"id":"T511","span":{"begin":168,"end":169},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"},{"id":"T512","span":{"begin":170,"end":182},"obj":"http://purl.obolibrary.org/obo/UBERON_0001981"},{"id":"T513","span":{"begin":170,"end":182},"obj":"http://www.ebi.ac.uk/efo/EFO_0000817"},{"id":"T514","span":{"begin":217,"end":222},"obj":"http://purl.obolibrary.org/obo/UBERON_0001638"},{"id":"T515","span":{"begin":217,"end":222},"obj":"http://www.ebi.ac.uk/efo/EFO_0000816"},{"id":"T516","span":{"begin":226,"end":234},"obj":"http://purl.obolibrary.org/obo/UBERON_0001637"},{"id":"T517","span":{"begin":226,"end":234},"obj":"http://www.ebi.ac.uk/efo/EFO_0000814"},{"id":"T518","span":{"begin":405,"end":421},"obj":"http://purl.obolibrary.org/obo/CL_0000115"},{"id":"T519","span":{"begin":543,"end":551},"obj":"http://purl.obolibrary.org/obo/UBERON_0000055"},{"id":"T520","span":{"begin":558,"end":559},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"}],"text":"A discussion of vascular disease should distinguish causal, pathological, structural and functional elements. The term “vasculopathy” represents any abnormality within a blood vessel- small, medium and large caliber, veins or arteries (reviewed in Seshan) [85]. Degenerative, metabolic and inflammatory conditions, embolic diseases and coagulation disorders are responsible for acute, subacute or chronic endothelial cell injury. In many cases, non-inflammatory (necrotizing) vascular injury causes accumulation of immune complexes within the vessel’s wall. A thickened intima with immunoglobulin and complement deposition, without inflammatory lymphocyte infiltration or atherosclerotic change are typical features of an autoimmune vasculopathy [86]."}

    LitCovid-PD-GO-BP

    {"project":"LitCovid-PD-GO-BP","denotations":[{"id":"T117","span":{"begin":336,"end":347},"obj":"http://purl.obolibrary.org/obo/GO_0050817"}],"text":"A discussion of vascular disease should distinguish causal, pathological, structural and functional elements. The term “vasculopathy” represents any abnormality within a blood vessel- small, medium and large caliber, veins or arteries (reviewed in Seshan) [85]. Degenerative, metabolic and inflammatory conditions, embolic diseases and coagulation disorders are responsible for acute, subacute or chronic endothelial cell injury. In many cases, non-inflammatory (necrotizing) vascular injury causes accumulation of immune complexes within the vessel’s wall. A thickened intima with immunoglobulin and complement deposition, without inflammatory lymphocyte infiltration or atherosclerotic change are typical features of an autoimmune vasculopathy [86]."}

    LitCovid-sentences

    {"project":"LitCovid-sentences","denotations":[{"id":"T267","span":{"begin":0,"end":109},"obj":"Sentence"},{"id":"T268","span":{"begin":110,"end":261},"obj":"Sentence"},{"id":"T269","span":{"begin":262,"end":429},"obj":"Sentence"},{"id":"T270","span":{"begin":430,"end":557},"obj":"Sentence"},{"id":"T271","span":{"begin":558,"end":751},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"A discussion of vascular disease should distinguish causal, pathological, structural and functional elements. The term “vasculopathy” represents any abnormality within a blood vessel- small, medium and large caliber, veins or arteries (reviewed in Seshan) [85]. Degenerative, metabolic and inflammatory conditions, embolic diseases and coagulation disorders are responsible for acute, subacute or chronic endothelial cell injury. In many cases, non-inflammatory (necrotizing) vascular injury causes accumulation of immune complexes within the vessel’s wall. A thickened intima with immunoglobulin and complement deposition, without inflammatory lymphocyte infiltration or atherosclerotic change are typical features of an autoimmune vasculopathy [86]."}

    LitCovid-PMC-OGER-BB

    {"project":"LitCovid-PMC-OGER-BB","denotations":[{"id":"T935","span":{"begin":170,"end":182},"obj":"UBERON:0001981"},{"id":"T936","span":{"begin":217,"end":222},"obj":"UBERON:0001638"},{"id":"T937","span":{"begin":226,"end":234},"obj":"UBERON:0001637"},{"id":"T938","span":{"begin":276,"end":285},"obj":"GO:0008152"},{"id":"T939","span":{"begin":336,"end":347},"obj":"GO:0050817"},{"id":"T940","span":{"begin":405,"end":416},"obj":"UBERON:0001986;CL:0000115"},{"id":"T941","span":{"begin":417,"end":421},"obj":"CL:0000115"},{"id":"T942","span":{"begin":515,"end":521},"obj":"UBERON:0002405"},{"id":"T943","span":{"begin":522,"end":531},"obj":"GO:0032991"},{"id":"T944","span":{"begin":543,"end":549},"obj":"UBERON:0000055"},{"id":"T945","span":{"begin":552,"end":556},"obj":"UBERON:0000309"},{"id":"T946","span":{"begin":582,"end":596},"obj":"GO:0019814"},{"id":"T947","span":{"begin":632,"end":644},"obj":"CL:0002629"},{"id":"T948","span":{"begin":645,"end":655},"obj":"CL:0000542"},{"id":"T949","span":{"begin":722,"end":732},"obj":"UBERON:0002405"},{"id":"T96879","span":{"begin":170,"end":182},"obj":"UBERON:0001981"},{"id":"T77733","span":{"begin":217,"end":222},"obj":"UBERON:0001638"},{"id":"T40084","span":{"begin":226,"end":234},"obj":"UBERON:0001637"},{"id":"T14710","span":{"begin":276,"end":285},"obj":"GO:0008152"},{"id":"T54940","span":{"begin":336,"end":347},"obj":"GO:0050817"},{"id":"T41404","span":{"begin":405,"end":416},"obj":"UBERON:0001986;CL:0000115"},{"id":"T37391","span":{"begin":417,"end":421},"obj":"CL:0000115"},{"id":"T70710","span":{"begin":515,"end":521},"obj":"UBERON:0002405"},{"id":"T76064","span":{"begin":522,"end":531},"obj":"GO:0032991"},{"id":"T45391","span":{"begin":543,"end":549},"obj":"UBERON:0000055"},{"id":"T89167","span":{"begin":552,"end":556},"obj":"UBERON:0000309"},{"id":"T23315","span":{"begin":582,"end":596},"obj":"GO:0019814"},{"id":"T46884","span":{"begin":632,"end":644},"obj":"CL:0002629"},{"id":"T48076","span":{"begin":645,"end":655},"obj":"CL:0000542"},{"id":"T55475","span":{"begin":722,"end":732},"obj":"UBERON:0002405"}],"text":"A discussion of vascular disease should distinguish causal, pathological, structural and functional elements. The term “vasculopathy” represents any abnormality within a blood vessel- small, medium and large caliber, veins or arteries (reviewed in Seshan) [85]. Degenerative, metabolic and inflammatory conditions, embolic diseases and coagulation disorders are responsible for acute, subacute or chronic endothelial cell injury. In many cases, non-inflammatory (necrotizing) vascular injury causes accumulation of immune complexes within the vessel’s wall. A thickened intima with immunoglobulin and complement deposition, without inflammatory lymphocyte infiltration or atherosclerotic change are typical features of an autoimmune vasculopathy [86]."}

    2_test

    {"project":"2_test","denotations":[{"id":"32700024-31227015-64991507","span":{"begin":747,"end":749},"obj":"31227015"}],"text":"A discussion of vascular disease should distinguish causal, pathological, structural and functional elements. The term “vasculopathy” represents any abnormality within a blood vessel- small, medium and large caliber, veins or arteries (reviewed in Seshan) [85]. Degenerative, metabolic and inflammatory conditions, embolic diseases and coagulation disorders are responsible for acute, subacute or chronic endothelial cell injury. In many cases, non-inflammatory (necrotizing) vascular injury causes accumulation of immune complexes within the vessel’s wall. A thickened intima with immunoglobulin and complement deposition, without inflammatory lymphocyte infiltration or atherosclerotic change are typical features of an autoimmune vasculopathy [86]."}