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{"target":"https://pubannotation.org/docs/sourcedb/PMC/sourceid/7323532","sourcedb":"PMC","sourceid":"7323532","source_url":"https://www.ncbi.nlm.nih.gov/pmc/7323532","text":"Patients infected with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) experience major lung damage due to viral replication and the ensuing cytokine storm and complex inflammatory processes (1). The severe damage to lung tissue can lead to secondary infections within a median of 17 days after onset of coronavirus disease (COVID-19) (2). Most immunocompetent patients who develop severe forms of COVID-19 have \u003e1 underlying condition, such as chronic obstructive pulmonary disease, hypertension, diabetes, or chronic kidney disease (3), but none of these predisposing factors generally are associated with an increased risk for developing fungal 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