PMC:7283670 / 125274-125488 JSONTXT

{"project":"LitCovid-PD-FMA-UBERON","denotations":[{"id":"T92","span":{"begin":31,"end":34},"obj":"Body_part"},{"id":"T93","span":{"begin":47,"end":51},"obj":"Body_part"}],"attributes":[{"id":"A92","pred":"fma_id","subj":"T92","obj":"http://purl.org/sig/ont/fma/fma24890"},{"id":"A93","pred":"fma_id","subj":"T93","obj":"http://purl.org/sig/ont/fma/fma12520"}],"text":"ACE2 is a prominent regulatory arm in the RAAS axis, thus, a disruption in ACE‐Angiotensin II‐Angiotensin Type 1 Receptor (AT1R), and ACE2/Angiotensin‐(1‐7)/Mas Receptor axes can result in multisystem inflammation."}

{"project":"LitCovid-PD-UBERON","denotations":[{"id":"T234","span":{"begin":31,"end":34},"obj":"Body_part"}],"attributes":[{"id":"A234","pred":"uberon_id","subj":"T234","obj":"http://purl.obolibrary.org/obo/UBERON_0001460"}],"text":"ACE2 is a prominent regulatory arm in the RAAS axis, thus, a disruption in ACE‐Angiotensin II‐Angiotensin Type 1 Receptor (AT1R), and ACE2/Angiotensin‐(1‐7)/Mas Receptor axes can result in multisystem inflammation."}

{"project":"LitCovid-PD-MONDO","denotations":[{"id":"T681","span":{"begin":201,"end":213},"obj":"Disease"}],"attributes":[{"id":"A681","pred":"mondo_id","subj":"T681","obj":"http://purl.obolibrary.org/obo/MONDO_0021166"}],"text":"ACE2 is a prominent regulatory arm in the RAAS axis, thus, a disruption in ACE‐Angiotensin II‐Angiotensin Type 1 Receptor (AT1R), and ACE2/Angiotensin‐(1‐7)/Mas Receptor axes can result in multisystem inflammation."}

{"project":"LitCovid-PD-CLO","denotations":[{"id":"T172","span":{"begin":8,"end":9},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"},{"id":"T173","span":{"begin":31,"end":34},"obj":"http://www.ebi.ac.uk/efo/EFO_0001410"},{"id":"T174","span":{"begin":59,"end":60},"obj":"http://purl.obolibrary.org/obo/CLO_0001020"}],"text":"ACE2 is a prominent regulatory arm in the RAAS axis, thus, a disruption in ACE‐Angiotensin II‐Angiotensin Type 1 Receptor (AT1R), and ACE2/Angiotensin‐(1‐7)/Mas Receptor axes can result in multisystem inflammation."}

{"project":"LitCovid-PD-CHEBI","denotations":[{"id":"T265","span":{"begin":79,"end":93},"obj":"Chemical"},{"id":"T266","span":{"begin":91,"end":93},"obj":"Chemical"},{"id":"T267","span":{"begin":94,"end":105},"obj":"Chemical"},{"id":"T2111","span":{"begin":139,"end":150},"obj":"Chemical"}],"attributes":[{"id":"A99410","pred":"chebi_id","subj":"T265","obj":"http://purl.obolibrary.org/obo/CHEBI_2719"},{"id":"A58585","pred":"chebi_id","subj":"T266","obj":"http://purl.obolibrary.org/obo/CHEBI_74067"},{"id":"A4493","pred":"chebi_id","subj":"T267","obj":"http://purl.obolibrary.org/obo/CHEBI_2719"},{"id":"A6603","pred":"chebi_id","subj":"T2111","obj":"http://purl.obolibrary.org/obo/CHEBI_2719"}],"text":"ACE2 is a prominent regulatory arm in the RAAS axis, thus, a disruption in ACE‐Angiotensin II‐Angiotensin Type 1 Receptor (AT1R), and ACE2/Angiotensin‐(1‐7)/Mas Receptor axes can result in multisystem inflammation."}

{"project":"LitCovid-PD-GO-BP","denotations":[{"id":"T19","span":{"begin":201,"end":213},"obj":"http://purl.obolibrary.org/obo/GO_0006954"}],"text":"ACE2 is a prominent regulatory arm in the RAAS axis, thus, a disruption in ACE‐Angiotensin II‐Angiotensin Type 1 Receptor (AT1R), and ACE2/Angiotensin‐(1‐7)/Mas Receptor axes can result in multisystem inflammation."}

{"project":"LitCovid-sentences","denotations":[{"id":"T1039","span":{"begin":0,"end":214},"obj":"Sentence"}],"namespaces":[{"prefix":"_base","uri":"http://pubannotation.org/ontology/tao.owl#"}],"text":"ACE2 is a prominent regulatory arm in the RAAS axis, thus, a disruption in ACE‐Angiotensin II‐Angiotensin Type 1 Receptor (AT1R), and ACE2/Angiotensin‐(1‐7)/Mas Receptor axes can result in multisystem inflammation."}

{"project":"LitCovid-PubTator","denotations":[{"id":"2592","span":{"begin":0,"end":4},"obj":"Gene"},{"id":"2593","span":{"begin":75,"end":78},"obj":"Gene"},{"id":"2594","span":{"begin":123,"end":127},"obj":"Gene"},{"id":"2595","span":{"begin":134,"end":138},"obj":"Gene"},{"id":"2604","span":{"begin":201,"end":213},"obj":"Disease"}],"attributes":[{"id":"A2592","pred":"tao:has_database_id","subj":"2592","obj":"Gene:59272"},{"id":"A2593","pred":"tao:has_database_id","subj":"2593","obj":"Gene:1636"},{"id":"A2594","pred":"tao:has_database_id","subj":"2594","obj":"Gene:185"},{"id":"A2595","pred":"tao:has_database_id","subj":"2595","obj":"Gene:59272"},{"id":"A2604","pred":"tao:has_database_id","subj":"2604","obj":"MESH:D007249"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"ACE2 is a prominent regulatory arm in the RAAS axis, thus, a disruption in ACE‐Angiotensin II‐Angiotensin Type 1 Receptor (AT1R), and ACE2/Angiotensin‐(1‐7)/Mas Receptor axes can result in multisystem inflammation."}