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PMC:7200337 / 20626-21074 JSONTXT

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2_test

Id Subject Object Predicate Lexical cue
32505227-28553293-46575269 171-175 28553293 denotes 2017
32505227-31140942-46575270 199-203 31140942 denotes 2019
32505227-32302401-46575271 354-358 32302401 denotes 2020
T5101 171-175 28553293 denotes 2017
T79329 199-203 31140942 denotes 2019
T2404 354-358 32302401 denotes 2020

LitCovid-PD-FMA-UBERON

Id Subject Object Predicate Lexical cue fma_id
T148 72-76 Body_part denotes lung http://purl.org/sig/ont/fma/fma7195
T149 100-112 Body_part denotes granulocytes http://purl.org/sig/ont/fma/fma62854
T150 250-260 Body_part denotes neutrophil http://purl.org/sig/ont/fma/fma62860
T151 273-283 Body_part denotes macrophage http://purl.org/sig/ont/fma/fma63261

LitCovid-PD-UBERON

Id Subject Object Predicate Lexical cue uberon_id
T16 72-76 Body_part denotes lung http://purl.obolibrary.org/obo/UBERON_0002048

LitCovid-PD-MONDO

Id Subject Object Predicate Lexical cue mondo_id
T143 116-124 Disease denotes SARS-CoV http://purl.obolibrary.org/obo/MONDO_0005091

LitCovid-PD-CLO

Id Subject Object Predicate Lexical cue
T169 72-76 http://purl.obolibrary.org/obo/UBERON_0002048 denotes lung
T170 72-76 http://www.ebi.ac.uk/efo/EFO_0000934 denotes lung
T171 153-157 http://purl.obolibrary.org/obo/CLO_0053428 denotes Camp

LitCovid-PD-GO-BP

Id Subject Object Predicate Lexical cue
T89 139-151 http://purl.obolibrary.org/obo/GO_0009405 denotes pathogenesis

LitCovid-PubTator

Id Subject Object Predicate Lexical cue tao:has_database_id
666 218-220 Gene denotes to Gene:6999
667 28-30 Gene denotes to Gene:6999
668 116-126 Species denotes SARS-CoV-2 Tax:2697049
669 261-268 Disease denotes NETosis

LitCovid-sentences

Id Subject Object Predicate Lexical cue
T116 0-205 Sentence denotes Lastly, more work is needed to ascertain the mechanistic role played by lung-resident and recruited granulocytes in SARS-CoV-2 control and pathogenesis (Camp and Jonsson, 2017, Flores-Torres et al., 2019).
T117 206-448 Sentence denotes In contrast to their early protective role, neutrophil NETosis and macrophage crosstalk can drive later-stage inflammatory cascades (Barnes et al., 2020), underscoring the overall pathogenic nature of damage-sensing host responses (Figure 2).