PMC:7161517 / 276-558 JSONTXT

{"project":"LitCovid-PD-FMA-UBERON","denotations":[{"id":"T2","span":{"begin":106,"end":110},"obj":"Body_part"}],"attributes":[{"id":"A2","pred":"fma_id","subj":"T2","obj":"http://purl.org/sig/ont/fma/fma12520"}],"text":"sregulation observed in SARS-CoV was previously postulated to be due to ACE2/angiotensin 1-7 (Ang1-7)/Mas axis downregulation; increased ACE2 activity was shown to mediate disease protection. Because angiotensin II receptor blockers, ACE inhibitors, and mineralocorticoid receptor a"}

{"project":"LitCovid-PD-MONDO","denotations":[{"id":"T5","span":{"begin":24,"end":32},"obj":"Disease"}],"attributes":[{"id":"A5","pred":"mondo_id","subj":"T5","obj":"http://purl.obolibrary.org/obo/MONDO_0005091"}],"text":"sregulation observed in SARS-CoV was previously postulated to be due to ACE2/angiotensin 1-7 (Ang1-7)/Mas axis downregulation; increased ACE2 activity was shown to mediate disease protection. Because angiotensin II receptor blockers, ACE inhibitors, and mineralocorticoid receptor a"}

{"project":"LitCovid-PD-CLO","denotations":[{"id":"T2","span":{"begin":142,"end":150},"obj":"http://purl.obolibrary.org/obo/CLO_0001658"}],"text":"sregulation observed in SARS-CoV was previously postulated to be due to ACE2/angiotensin 1-7 (Ang1-7)/Mas axis downregulation; increased ACE2 activity was shown to mediate disease protection. Because angiotensin II receptor blockers, ACE inhibitors, and mineralocorticoid receptor a"}

{"project":"LitCovid-PD-CHEBI","denotations":[{"id":"T3","span":{"begin":77,"end":88},"obj":"Chemical"},{"id":"T4","span":{"begin":200,"end":214},"obj":"Chemical"},{"id":"T7","span":{"begin":200,"end":211},"obj":"Chemical"},{"id":"T8","span":{"begin":212,"end":214},"obj":"Chemical"},{"id":"T9","span":{"begin":234,"end":248},"obj":"Chemical"},{"id":"T10","span":{"begin":238,"end":248},"obj":"Chemical"},{"id":"T11","span":{"begin":254,"end":271},"obj":"Chemical"}],"attributes":[{"id":"A3","pred":"chebi_id","subj":"T3","obj":"http://purl.obolibrary.org/obo/CHEBI_48433"},{"id":"A4","pred":"chebi_id","subj":"T4","obj":"http://purl.obolibrary.org/obo/CHEBI_2719"},{"id":"A5","pred":"chebi_id","subj":"T4","obj":"http://purl.obolibrary.org/obo/CHEBI_48432"},{"id":"A6","pred":"chebi_id","subj":"T4","obj":"http://purl.obolibrary.org/obo/CHEBI_58506"},{"id":"A7","pred":"chebi_id","subj":"T7","obj":"http://purl.obolibrary.org/obo/CHEBI_48433"},{"id":"A8","pred":"chebi_id","subj":"T8","obj":"http://purl.obolibrary.org/obo/CHEBI_74067"},{"id":"A9","pred":"chebi_id","subj":"T9","obj":"http://purl.obolibrary.org/obo/CHEBI_35457"},{"id":"A10","pred":"chebi_id","subj":"T10","obj":"http://purl.obolibrary.org/obo/CHEBI_35222"},{"id":"A11","pred":"chebi_id","subj":"T11","obj":"http://purl.obolibrary.org/obo/CHEBI_25354"}],"text":"sregulation observed in SARS-CoV was previously postulated to be due to ACE2/angiotensin 1-7 (Ang1-7)/Mas axis downregulation; increased ACE2 activity was shown to mediate disease protection. Because angiotensin II receptor blockers, ACE inhibitors, and mineralocorticoid receptor a"}

{"project":"LitCovid-PMC-OGER-BB","denotations":[{"id":"T10","span":{"begin":24,"end":32},"obj":"SP_10"},{"id":"T11","span":{"begin":72,"end":76},"obj":"G_3;PG_10;PR:000003622"},{"id":"T12","span":{"begin":77,"end":90},"obj":"PR:000036013"},{"id":"T13","span":{"begin":94,"end":98},"obj":"PR:000036013"},{"id":"T14","span":{"begin":137,"end":141},"obj":"G_3;PG_10;PR:000003622"},{"id":"T15","span":{"begin":200,"end":214},"obj":"DG_37;PR:000001118"},{"id":"T16","span":{"begin":215,"end":223},"obj":"PR:000001118"},{"id":"T17","span":{"begin":238,"end":248},"obj":"CHEBI:35222;CHEBI:35222"},{"id":"T18","span":{"begin":254,"end":271},"obj":"CHEBI:25354;CHEBI:25354"},{"id":"T31949","span":{"begin":24,"end":32},"obj":"SP_10"},{"id":"T17436","span":{"begin":72,"end":76},"obj":"G_3;PG_10;PR:000003622"},{"id":"T37458","span":{"begin":77,"end":90},"obj":"PR:000036013"},{"id":"T46294","span":{"begin":94,"end":98},"obj":"PR:000036013"},{"id":"T60166","span":{"begin":137,"end":141},"obj":"G_3;PG_10;PR:000003622"},{"id":"T39187","span":{"begin":200,"end":214},"obj":"DG_37;PR:000001118"},{"id":"T33501","span":{"begin":215,"end":223},"obj":"PR:000001118"},{"id":"T28542","span":{"begin":238,"end":248},"obj":"CHEBI:35222;CHEBI:35222"},{"id":"T14634","span":{"begin":254,"end":271},"obj":"CHEBI:25354;CHEBI:25354"}],"text":"sregulation observed in SARS-CoV was previously postulated to be due to ACE2/angiotensin 1-7 (Ang1-7)/Mas axis downregulation; increased ACE2 activity was shown to mediate disease protection. Because angiotensin II receptor blockers, ACE inhibitors, and mineralocorticoid receptor a"}

{"project":"LitCovid-PubTator","denotations":[{"id":"23","span":{"begin":72,"end":92},"obj":"Gene"},{"id":"24","span":{"begin":94,"end":100},"obj":"Gene"},{"id":"25","span":{"begin":137,"end":141},"obj":"Gene"},{"id":"26","span":{"begin":234,"end":237},"obj":"Gene"},{"id":"27","span":{"begin":254,"end":280},"obj":"Gene"},{"id":"33","span":{"begin":24,"end":32},"obj":"Species"}],"attributes":[{"id":"A23","pred":"tao:has_database_id","subj":"23","obj":"Gene:59272"},{"id":"A24","pred":"tao:has_database_id","subj":"24","obj":"Gene:284"},{"id":"A25","pred":"tao:has_database_id","subj":"25","obj":"Gene:59272"},{"id":"A26","pred":"tao:has_database_id","subj":"26","obj":"Gene:1636"},{"id":"A27","pred":"tao:has_database_id","subj":"27","obj":"Gene:4306"},{"id":"A33","pred":"tao:has_database_id","subj":"33","obj":"Tax:694009"}],"namespaces":[{"prefix":"Tax","uri":"https://www.ncbi.nlm.nih.gov/taxonomy/"},{"prefix":"MESH","uri":"https://id.nlm.nih.gov/mesh/"},{"prefix":"Gene","uri":"https://www.ncbi.nlm.nih.gov/gene/"},{"prefix":"CVCL","uri":"https://web.expasy.org/cellosaurus/CVCL_"}],"text":"sregulation observed in SARS-CoV was previously postulated to be due to ACE2/angiotensin 1-7 (Ang1-7)/Mas axis downregulation; increased ACE2 activity was shown to mediate disease protection. Because angiotensin II receptor blockers, ACE inhibitors, and mineralocorticoid receptor a"}